Figure 1. Exemplars of Human-Computer Integration: extending the body with additional robotic arms; [70] embedding computation into the body using electric muscle stimulation to manipulate handwriting [48]; and, a tail extension controlled by body movements [86].

This paper explores how microgestures can allow us to execute a secondary task, for example controlling mobile applications, without interrupting the manual primary task, for instance, driving a car. In order to design microgestures iteratively, we interviewed sports-and physiotherapists while asking them to use task related props, such as a steering wheel, a cash card , and a pen for simulating driving a car, an ATM scenario, and a drawing task. The primary objective here is to define microgestures that are easily performable without interrupting or interfering the primary task. Using expert interviews, we developed a taxonomy that classifies these gestures according to their task context. We also assessed the ergonomic and attentional attributes that influence the feasibility and task suitability of microinteractions, and evaluated their level of resources required. Accordingly, we defined 21 microgestures that allow performing microinteractions within a manual, dual task context. Our taxonomy poses a basis for designing microinteraction techniques.

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Lipoprotein(a), as an atherogenic particle, represents an independent risk factor for coronary heart disease. In the present study the morphological distribution of apoprotein (a) and apoprotein B within the arterial wall is described. Apoprotein B, a constituent of very low-density lipoprotein, low-density lipoprotein and lipoprotein(a) has previously been demonstrated in atheromatous lesions. Lipoprotein(a) possesses an additional protein, designated apoprotein (a). Autopsy material (n = 74) from the left coronary artery and from the thoracic aorta has been examined by means of immunohistochemistry and both apoprotein (a) and apoprotein B were detected, primarily associated with the extracellular matrix and accumulating in lesions in the arterial wall. The staining pattern for both antigens was almost always found to be congruent, suggesting that the detection of (a)-antigen has to be attributed at least in part to the presence of lipoprotein(a). It is concluded that both low-density lipoprotein and lipoprotein(a) have an important role in the pathogenesis of atherosclerosis.

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