Thyroid-stimulating immunoglobulins in neonatal Graves's disease.

Teng, C.S.; Tong, Tony C.; Hutchison, J H; Yeung, Raymond W.

doi:10.1136/adc.55.11.894

Search citation statements

Order By: Relevance

Paper Sections

Select...

Discussion1

Citation Types

Supporting

Mentioning

Contrasting

Year Published

1983

2012

Publication Types

Select...

Article5

Book1

Relationship

Self Cite0

Independent6

Authors

Journals

Cited by 7 publications

(1 citation statement)

References 6 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…The exposure of fetal thyroid gland to thyroid-stimulating immunoglobulin in utero can cause hyperthyroidism. 10,11 Fetal thyrotoxicosis is manifested by tachycardia and hyperkinesia and may be complicated by congestive heart failure. 12,13 In heart failure, chylothorax may be produced by an increase in lymph production secondary to an augmented pulmonary capillary filtration associated with an obstruction to lymphatic flow caused by increased pressure in the left subclavian vein.…”

Section: Discussionmentioning

confidence: 99%

Congenital Chylothorax in Neonatal Thyrotoxicosis

et al. 1999

View full text Add to dashboard Cite

We report a patient with congenital chylothorax who also had neonatal thyrotoxicosis secondary to maternal Graves' disease. Fetal tachycardia with hydrops was detected at 28 weeks' gestational age. The fetus responded to antithyroid medication in utero but had persistent bilateral pleural effusion. At birth, he had respiratory distress due to massive pleural effusion. Cytologic studies of pleural fluid were consistent with chylothorax. To the best of our knowledge, the association of congenital chylothorax with fetal (neonatal) thyrotoxicosis, has not been reported previously.

show abstract

Section: Discussionmentioning

confidence: 99%