Respiratory allergy due to chemical compounds encountered in the rubber, lacquer, shellac, and beauty culture industries

Gelfand, H.Harold

doi:10.1016/0021-8707(63)90056-x

Cited by 74 publications

(28 citation statements)

References 10 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…Many of the cases were proven by specific challenge tests. Several nonantibiotic drugs can also cause occupational asthma, such as tylosin tartrate [123], alpha-methyldopa [124], amprolium hydrochloride [125], and morphine [126].…”

Section: Pharmaceutical Productsmentioning

confidence: 99%

Aetiological agents in occupational asthma

Chan‐Yeung¹,

Malo²

1994

Eur Respir J

210

111

View full text Add to dashboard Cite

A Ae et ti io ol lo og gi ic ca al l a ag ge en nt ts s i in n o oc cc cu up pa at ti io on na al l a as st th hm ma a M. Chan-Yeung*, J-L. Malo** Aetiological agents in occupational asthma. M. Chan-Yeung, J-L. Malo. ERS Journals Ltd 1994. ABSTRACT: Occupational asthma has become the most prevalent occupational lung disease in developed countries. At present, about 200 agents have been implicated in causing occupational asthma in the workplace. These agents can be divided into two categories by their mechanism of action: immunological and nonimmunological.Immunological causes can be further divided into those that induce asthma through an immunoglobulin E (IgE)-dependent mechanism, and those that induce asthma through a non-IgE-dependent mechanism. In the latter category, specific IgE antibodies are found only in a small percentage of the patients with proven disease, even though the clinical picture is compatible with an allergic reaction. The immunological mechanism(s) responsible for these agents has yet to be identified.The best known example of nonimmunological asthma is Reactive Airways Dysfunction Syndrome (RADS) or irritant-induced asthma.In this review, examples of types of agents causing occupational asthma are discussed and a compendium table of aetiological agents is given. Eur Respir J., 1994, 7, 346-371.

show abstract

Section: Pharmaceutical Productsmentioning

confidence: 99%

Aetiological agents in occupational asthma

Chan‐Yeung¹,

Malo²

1994

Eur Respir J

210

111

View full text Add to dashboard Cite

show abstract

“…Hairdressers are exposed to a variety of irritative and allergenic substances,2 which can cause airway symptoms and diseases related to work.3 4Hair lacquers and permanent wave solutions can irritate airways and worsen the symptoms of people with reactive airways or asthma.5'-Several case reports have been published on occupational asthma caused by persulphate salts in hair bleaches. '2 16 Henna can also cause asthma.1'17 Polyvinylpyrrolidone in hair lacquers has been suspected of causing alveolitis"' '9 and lung granulomatosis (thesaurosis),20 21 but the results have been controversial and upon closer scrutiny the association is weak or lacking.22 23 Our aim was to study the self reported prevalence of respiratory symptoms and diseases, and to estimate their risk among hairdressers compared with saleswomen.…”

mentioning

confidence: 99%

Self reported respiratory symptoms and diseases among hairdressers.

et al. 1997

View full text Add to dashboard Cite

Objectives-Hairdressers are exposed to many irritative and allergenic substances capable of causing occupational respiratory symptoms and diseases. The self reported prevalence of respiratory symptoms and diseases was studied, and the risks among hairdressers compared with saleswomen was estimated. Methods-A cross sectional prevalence study of respiratory symptoms and diseases was carried out among hairdressers and supermarket saleswomen, with a computer assisted telephone interview method (CATI). The These were used to define chronic bronchitis, and asthma, laryngitis, and allergic rhinitis diagnosed by a physician. Results-There was a considerable difference in the prevalence of chronic bronchitis; 6.8% in hairdressers versus 1.9% in saleswomen. The odds ratio (OR) adjusted for age, smoking, and atopy for chronic bronchitis indicated an increased risk of chronic bronchitis (OR 4.8, 95% confidence interval (95% CI) 2.2 to 10.1). No association was found between work as a hairdresser and asthma, laryngitis, and allergic rhinitis. Also the prevalence of rhinitis, rhinitis with eye symptoms, cough with phlegm, dyspnoea, and dyspnoea accompanied by cough was increased among hairdressers. The corresponding adjusted risk ORs were 1.7 (95% CI 1.3 to 2.3) for rhinitis, 1.9 (95% CI 1.4 to 2.6) for rhinitis with eye symptoms, 1.4 (CI 1.1 to 1.9) for cough with phlegm, 1.5 (95% CI 1.0 to 2.2) for dyspnoea, and 1.6 (95% CI 1.0 to 2.7) for dyspnoea with cough.Conclusions-Our results indicate an increased prevalence of upper and lower respiratory symptoms among hairdressers. Allergenic and irritative chemicals in hairdressing are likely candidates explaining the difference found between the hairdressers and controls. Work related reasons should be considered when a hairdresser presents with airway symptoms. Preventive actions are needed to improve the working conditions and personal protection. (Occup Environ Med 1997;54:452-455)

show abstract

“…Dernehl et al (14) provided the first description of OA due to ethylenediamine in persons working in the epoxy resin industry. Using a SIC, Gelfand et al (15) implicated this agent in the occurrence of OA in persons employed in manufacture of shellac. The mechanism by which ethylenediamine causes asthma is not well-defined (12).…”

Section: Discussionmentioning

confidence: 99%