“…Inhibit extracellular mediators that may enhance inflammatory or fibrotic processes Anti-TGF-b1 and TGF-b2 antibodies deceased adhesion formation in rats and mice [235,236] Anti-TGF-b1 antibody reduced cellularity of adhesions in rats, not incidence [237] Monotherapy with anti-IL-1 antibody decreased adhesions in rat model [238] Therapy with anti-IL-1 and anti-TNF-a antibodies decreased adhesions in rat model; effect superior to either monotherapy [238] sunitinib (vascular endothelial growth factor receptor 2 antagonist) reduced adhesion formation in mice [239] Epidermal growth factor Possibly increase cell proliferation and accelerate healing process Some efficacy in rat model [240] Did not affect hydroxyproline content of tissue [240] Angiotensin-converting enzyme inhibitors (ACE-I) (Benazepril, captopril, enalapril, fosinopril, lisinopril, moexipril, quinapril, ramipril)…”