Pulmonary strongyloidiasis: an unusual opportunistic pneumonia in a patient with AIDS.

Makris, A N; Шер, С. А.; Bertoli, Ciro João; Latour, Maëlle

doi:10.2214/ajr.161.3.8352101

Cited by 27 publications

(12 citation statements)

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“…Use of corticosteroids, even for short durations, for underlying chronic lung disease or other indications is a risk factor for the development of hyperinfection syndrome with pulmonary involvement (25,44,49,60,194). Though rare, pulmonary strongyloidiasis is a potential complication of HIV/AIDS (43,51,97,101). Several other factors, such as hematologic malignancy, immunosuppressive drug therapy, solid organ transplantation, hematopoietic cell transplantation, human T-cell leukemia virus type 1 (HTLV-1) infection, hypogammaglobulinemia, and malnutrition, have all also been associated with Strongyloides hyperinfection syndrome (79,164,197).…”

Section: Strongyloidesmentioning

confidence: 99%

Eosinophilic Pneumonias

2012

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SUMMARY This review starts with discussions of several infectious causes of eosinophilic pneumonia, which are almost exclusively parasitic in nature. Pulmonary infections due specifically to Ascaris , hookworms, Strongyloides , Paragonimus , filariasis, and Toxocara are considered in detail. The discussion then moves to noninfectious causes of eosinophilic pulmonary infiltration, including allergic sensitization to Aspergillus , acute and chronic eosinophilic pneumonias, Churg-Strauss syndrome, hypereosinophilic syndromes, and pulmonary eosinophilia due to exposure to specific medications or toxins.

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Section: Strongyloidesmentioning

confidence: 99%

Eosinophilic Pneumonias

2012

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“…In contrast, in immunocompromised hosts, e.g., patients with acquired immunode®ciency syndrome (AIDS; Makris et al 1993) or malignancy (Nandy et al 1995) and individuals receiving corticosteroids (Liu and Weller 1993;Woodring et al 1994), disseminated pulmonary hyperinfection can develop due to accelerated reproduction by the parasite via internal autogenous reinfection, i.e., autoinfection (Mans®eld et al 1995). The pulmonary disseminated disease carries a mortality rate of nearly 80% (Igra- Siegman et al 1981;Simpson et al 1993).…”

Section: Introductionmentioning

confidence: 99%

Strongyloides stercoralis : identification of antigens in natural human infections from endemic areas of the United States

Siddiqui

Koenig

Sinensky

et al. 1997

Parasitology Research

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Using Western-blot analysis, we identified eight immunodominant antigens (apparent molecular weights 96, 86, 75, 56, 41, 32, 28, and 26 kDa) of Strongyloides stercoralis in natural human infections. For this study, 78 individual serum samples were obtained from S. stercoralis-infected patients residing in endemic areas of the United States. Poly A+ RNA was translated in vitro in the rabbit-reticulocyte lysate system. The newly synthesized translation products were immuno-precipitated with S. stercoralis human infection sera. All eight of the identified antigens were detected in the immunoprecipitates. The potential of these antigens as targets for immunodiagnosis is also discussed.

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“…It can also cause hyperinfection syndrome or disseminated disease in immunocompromised patients (Kramer et al, 1990). However, disseminated infection of Strongyloides stercoralis to the lung is extremely rare (Armignacco et al, 1989;Glezerov & Masci, 1990;Gomples et al, 1991;Makris et al, 1993). The common symptoms and signs are dyspnea, cough, sputum, wheezing and hemoptysis.…”

Section: Other Parasitesmentioning

confidence: 99%