A 35-year-old male patient with palpitations and mild lightheadedness was admitted to our clinic. Short-lasting paroxysmal ventricular tachycardia was diagnosed following 12-lead electrocardiography (ECG). A mass that included two-thirds of the interventricular septum and the left ventricular cavity was seen by two-dimensional echocardiography and magnetic resonance imaging (MRI). Specific hemagglutination tests for hydatid cyst were positive. The mass was excised, and the patient had a septoplasty operation to repair the remaining septal defect. He is currently being followed and reports no complaints. No evidence of arrhythmia was noted in 24-hour ambulatory ECG monitoring.

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Most cardiac aneurysms develop after myocardial infarction. Calcification in the aneurysmal wall is seen rarely. In this case report the authors present a thirty-nine-year-old man, who had been free from symptoms until eight months before, when he began to experience palpitations due to monomorphic sustained ventricular tachycardia. A chest roentgenogram disclosed an oval calcification on the left ventricular apex. Coronary angiography and left ventriculography revealed normal epicardial coronary arteries and a massive calcified and ossified left ventricular apical aneurysm. He had no chest pain, nor were there electrocardiographic findings of myocardial infarction. Attacks of ventricular tachycardia disappeared after aneurysmectomy was performed. To the author's knowledge there is no case report in the literature of a calcified left ventricular aneurysm with normal epicardial coronary arteries and without clinical and electrocardiographic findings of infarction. They discuss the possible etiology of this case.

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Many types of coronary artery anomalies have been detected, and most of them occur in the left circumflex artery. In this report a unique circumflex artery anomaly is presented. The left circumflex artery arose as a terminal extension of the right coronary artery. The main stem and obtuse marginal branches of the circumflex artery were normal and well developed.

Lipids, lipoproteins and apolipoproteins are among the risk factors for the most serious health problem of the age--coronary artery disease (CAD). They vary from country to country, from area to area within a country, depending on genetic, environmental, dietary and many other factors. Our aim was to determine the levels of lipids, lipoproteins and apolipoproteins in healthy people in the central Black Sea region of Turkey. Subjects included 1348 volunteers (682 women, 666 men) referred to the Medical Faculty hospital from the study area. The population consisted of healthy people or those whose disease was not affecting the metabolism of lipids. Cholesterol, triglyceride and HDL-cholesterol levels in the obtained serum samples were measured spectrophotometrically, while apolipoprotein A-I, apolipoprotein B, apolipoprotein E and lipoprotein(a) levels were measured nephelometrically. The levels of lipid parameters were as follows: total cholesterol for men was 4.22 +/- 1.00 mmol/l (mean arithmetic +/- SD), triglyceride 1.20 mmol/l (0.30-4.44) [geometric mean (range)], HDL-cholesterol 0.88 +/- 0.22 mmol/l, LDL-cholesterol 2.69 +/- 0.85 mmol/l, apolipoprotein A-I 1.26 +/- 0.22 mmol/l apolipoprotein B 1.12 +/- 0.32 mmol/l, apolipoprotein E 0.037 +/- 0.012 mmol/l and lipoprotein(a) 0.25 g/l (0.03-2.75); total cholesterol for women was 4.53 +/- 1.00 mmol/l, triglyceride 1.05 mmol/l (0.28-4.50), HDL-cholesterol 1.08 +/- 0.26 mmol/l, LDL-cholesterol 2.87 +/- 0.88 mmol/l, apolipoprotein A-I 1.45 +/- 0.25 mmol/l, apolipoprotein B 1.11 +/- 0.31 mmol/l, apolipoprotein E 0.039 +/- 0.011 mmol/l and lipoprotein(a) 0.22 g/l (0.03 2.16). In conclusion, our study in four different regions in Turkey reflected that the people living in the central Black Sea region are less vulnerable to the risk of CAD, although at a relatively higher risk compared to some other countries.

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