Salmonella enterica success on infection and transmission between hosts relies greatly on its ability to recognize, to evade, and to even exploit host defenses on its favor (Behnsen et al., 2015). Critical to Salmonella pathogenesis is the coordinated assembly of two type III secretion systems (T3SS) that are located at specific genome regions called pathogenicity islands (SPIs). The Salmonella pathogenicity island SPI-1 is associated with the invasion of epithelial cells, whereas SPI-2 is required for intracellular survival and proliferation (Agbor &
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