<strong style="text-align: justify;"><span style="font-size: 10pt; font-family: 'Times New Roman', serif;">Introdução</span></strong><span style="text-align: justify; font-size: 10pt; font-family: 'Times New Roman', serif;">: Após a implantação do Programa Nacional de Atenção à Saúde Auditiva (PNASA), em 2004, houve incontestáveis avanços na assistência ao deficiente auditivo. Porém, existem divergências na execução desse programa que dificultam o seu progresso e limitam o acesso à Saúde Auditiva. Tais evidências ilustram a necessidade da criação de uma rotina para avaliá-lo a fim de reorientar e monitorar as práticas dos atores envolvidos. <strong>Objetivo:</strong> Analisar os progressos e entraves da Política de Saúde Auditiva do país na atual conjuntura brasileira. <strong>Metodologia</strong>: Trata-se de um estudo exploratório com abordagem quantitativa realizado no período de junho a agosto de 2013, através da análise de dados secundários (SAI/SUS e CNES). <strong>Conclusão</strong>: Observou-se que a implantação do PNASA não é uniforme no país. A disparidade está evidenciada na cobertura nacional e distribuição local desse serviço, o que denota um imenso contingente de deficientes auditivos não contemplados com a assistência à Saúde Auditiva, ferindo os princípios de integralidade e universalidade do SUS.</span>

SNHL is a common complication in SCA; furthermore, this study identified a significant association between ED and SNHL. Damage to the vascular endothelium because of inflammation in SCA reduced blood flow in the inner ear. Thus, this circulatory disorder culminates in vaso-occlusive process and induces auditory disorders, such as SNHL.

The workings of the auditory pathway of patients with tinnitus and normal hearing can be associated with an auditory efferent pathway dysfunction at the level of the superior olivary complex. Otoacoustic emission suppression with contralateral noise can represent an alternative to its evaluation. Aim: to investigate Transient Otoacoustic Emission suppression in normal hearing adults with and without tinnitus and to compare the two groups. Study design: cross-sectional contemporary cohort. Materials and Methods: we assessed 40 female individuals between 18 and 59 years of age, 20 without tinnitus and 20 with it. We studied the TOAE suppression with a contralateral white noise at 50 dBSPL. Results: TOAE amplitude was lower in the group with tinnitus. There was no difference between the groups with and without tinnitus in terms of TOAE suppression, except in the frequency of 1000 Hz in the left ear in the tinnitus group. Conclusion: the afferent system assessment may contribute to the topographic diagnosis of tinnitus; however, we still need further studies to establish the proper methodology and normative values to carry out these tests.

Endocochlear, retrocochlear and/or central origin hearing damage may be related to the absence of appropriate levels of thyroid hormone during morphogenesis and/or auditory system development. Hearing disorders related to the thyroid are not well studied, despite speculation on the pathophysiological mechanisms. The objective of this review was to characterize the main pathophysiological mechanisms of congenital hypothyroidism and to evaluate the relationship with central and peripheral hearing disorders. We conducted a literature review using the databases MedLine, LILACS, Cochrane Library, SciELO, Institute for Scientific Information (ISI), Embase, and Science Direct between July and September on 2016. We identified the studies that address hearing disorder mechanisms on the congenital hypothyroidism. Congenital hypothyroidism may have clinical and subclinical manifestations that affect the auditory system and may be a potential risk factor for hearing impairment. Hearing impairment can severely impact quality-of-life, which emphasizes the importance of monitoring and evaluating hearing during the clinical routine of these patients. Arch Endocrinol Metab. 2017;61(5):501-5