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gastric hyperplastic polyps, adenocarcinomas of the distal part of the stomach, and lymphomas of mucosa-associated lymphoid tissue. [1,2] Colonization of stomach by H. pylori and chronic active gastritis present a cause-and-effect relationship. Healing of gastric inflammatory lesions using eradication therapy, as a remedial measure of gastric inflammatory lesions may be deficient in immediate clinical benefits; however, it yields positive results such as thearrest and reversal of gastric histological lesions, and long term consequences such as reduction in gastric atrophy and intestinal metaplasia, which are precursors of gastric carcinoma. [3] Methods of detection are divided into invasive and noninvasive tests. Noninvasive tests include serology and carbon-labeled urea breath test. The invasive tests include the rapid urease test, histological examination, and culture. Among the invasive tests,