A two year old male Labrador Retriever was treated with delmadinone acetate because of benign prostatic hyperplasia. Four days after the injection the dog showed gastrointestinal signs and a progressive lethargy. In the hospital for small animals of the Justus-Liebig-University of Gießen an ACTH stimulation test was done and a secondary hypoadrenocorticism was diagnosed. The dog was treated with prednisolone in physiological dose for 14 weeks after the injection. The clinical symptoms stopped immediately. A new ACTH stimulation test some weeks later showed a completely normal adrenal function.
A 15-month-old female intact Australian shepherd dog was presented with a history of acute, frequent vomiting and progressive lethargy. Only a painful abdomen was detected on physical examination. Laboratory results were suggestive of inflammation. Markedly thickened gastric mucosa with loss of gastric wall layering next to enlarged gastric lymph nodes and signs of peritonitis were detected on abdominal ultrasound. Explorative laparotomy revealed severe enlargement of the stomach wall thickness and gastric lymph nodes. High-grade T-cell lymphoma was diagnosed on histopathology and cytology. Despite the poor prognosis, a combination chemotherapy was initiated. During therapy, the dog had a good quality of life, apart from mild side effects of chemotherapeutic drugs. Neoplastic diseases such as alimentary lymphoma are rare in young dogs. With 252 days of survival, this is the youngest dog with long-term survival of a primarily alimentary T-cell lymphoma of the stomach, showing that appropriate chemotherapy can have a favourable outcome.
Two dogs with increased thyroxin concentrations compatible with hyperthyroidism were referred for further examinations. One dog displayed clinical signs of hyperthyroidism. Based on history, clinical examination, laboratory evaluation and scintigraphy an alimentary thyrotoxicosis was identified. It was caused by feeding a BARF diet containing thyroidal tissue in one dog and by conventional dog food in the other patient. After changing the diet the clinical signs resolved in the affected dog. A control examination revealed thyroxin concentrations within the reference range in both dogs.
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