The effect caffeine elicits on endurance performance is well founded. However, comparatively less research has been conducted on the ergogenic potential of anaerobic performance. Some studies showing no effect of caffeine on performance used untrained subjects and designs often not conducive to observing an ergogenic effect. Recent studies incorporating trained subjects and paradigms specific to intermittent sports activity support the notion that caffeine is ergogenic to an extent with anaerobic exercise. Caffeine seems highly ergogenic for speed endurance exercise ranging in duration from 60 to 180 seconds. However, other traditional models examining power output (i.e. 30-second Wingate test) have shown minimal effect of caffeine on performance. Conversely, studies employing sport-specific methodologies (i.e. hockey, rugby, soccer) with shorter duration (i.e. 4-6 seconds) show caffeine to be ergogenic during high-intensity intermittent exercise. Recent studies show caffeine affects isometric maximal force and offers introductory evidence for enhanced muscle endurance for lower body musculature. However, isokinetic peak torque, one-repetition maximum and muscular endurance for upper body musculature are less clear. Since relatively few studies exist with resistance training, a definite conclusion cannot be reached on the extent caffeine affects performance. It was previously thought that caffeine mechanisms were associated with adrenaline (epinephrine)-induced enhanced free-fatty acid oxidation and consequent glycogen sparing, which is the leading hypothesis for the ergogenic effect. It would seem unlikely that the proposed theory would result in improved anaerobic performance, since exercise is dominated by oxygen-independent metabolic pathways. Other mechanisms for caffeine have been suggested, such as enhanced calcium mobilization and phosphodiesterase inhibition. However, a normal physiological dose of caffeine in vivo does not indicate this mechanism plays a large role. Additionally, enhanced Na+/K+ pump activity has been proposed to potentially enhance excitation contraction coupling with caffeine. A more favourable hypothesis seems to be that caffeine stimulates the CNS. Caffeine acts antagonistically on adenosine receptors, thereby inhibiting the negative effects adenosine induces on neurotransmission, arousal and pain perception. The hypoalgesic effects of caffeine have resulted in dampened pain perception and blunted perceived exertion during exercise. This could potentially have favourable effects on negating decreased firing rates of motor units and possibly produce a more sustainable and forceful muscle contraction. The exact mechanisms behind caffeine's action remain to be elucidated.

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This study compared respiratory compensation thresholds (RCT) ( VCO(2) inflection point) of competitors in highly aerobic events (aerobic competitors, ARC) ( n=16), competitors in highly anaerobic events (anaerobic competitors, ANC) ( n=15), and untrained subjects (UT) ( n=25). Maximal oxygen consumption ( VO(2max)), respiratory compensation threshold as a percentage of VO(2max) (RCT), and VO(2) at RCT ( Vdot;O(2RCT)) were determined during a maximal Bruce treadmill protocol. VO(2max) (ml x kg(-1) min(-1)) was significantly greater ( P<0.05) for ARC [67.2 (8.5)] than for ANC [50.0 (7.8)] and UT [43.8 (5.4)]. However, the difference between ANC and UT only approached significance ( P=0.07). RCT was not significantly different between ARC [76.3 (8.7)] and ANC [80.7 (6.8)] but was significantly lower ( P<0.05) for UT [62.5 (8.8)]. VO(2RCT) (ml x kg(-1) min(-1)) was significantly greater ( P<0.05) for ARC [51.6 (11.0)] and ANC [40.2 (6.6)] than for UT [27.4 (5.4)], with a significant difference also between ARC and ANC. While used as a criterion for group assignment, greater VO(2max), as well as RCT values in ARC (vs UT), reflect chronic aerobic training adaptations. ANC demonstrated VO(2max) values intermediate to ARC and UT, with RCT very comparable to those found in ARC. The results suggest subjects competitive in highly anaerobic events do not possess excessively high VO(2max) values. These individuals, however, demonstrate a high RCT when values are expressed relative to VO(2max). Oxygen consumption at the RCT in this group is superior to that in UT but inferior to that in ARC, which likely has important implications regarding performance.

Nineteen physically active men supplemented their diet with 20 g per day creatine monohydrate (Cr group) or placebo (PI group) for 6 days. Before and after supplementation, subjects performed 3 arm Wingates (AW1, AW2, and AW3) and 3 leg Wingates (LW1, LW2, and LW3) on consecutive days. Wingates were separated by 2 minutes each. Mean power (MP), peak power (PP), and percent decrease (%D) were compared between and within groups. MP did not change significantly for arms or legs. PP did not change significantly for legs. PP increased significantly in the Cr group (AW1) and for the P1 group (AW1 and AW3). MP and PP were not significantly different between groups. The %D increased significantly in the P1 group (AW1, AW3, and LW3). For the Cr group, %D decreased significantly (pre vs. post) and was significantly lower than for the P1 group (LW2-post). Results suggest that short-term Cr supplementation does not enhance MP and PP during repeated upper- and lower-body Wingate tests when not accompanied by an increase in body weight. However, changes in %D suggest possible ergogenic effects.

A wheelchair frame-mounted accelerometer differentiated between perceptually-prescribed low and moderate effort levels and may prove to be a valid instrument in the detection of a wheelchair users' physical activity. [Box: see text].

This study examined heat stress, heart rate (HR), fluid balance, micro-environment temperature and humidity with Islamic athletic clothing (IC) compared to traditional soccer uniform (SC). Ratings of perceived exertion (RPE), session RPE (S-RPE), comfort, and cooling response were also examined. Female volunteers (N = 8) completed a treadmill [Formula: see text] test and then, in a randomized, counter-balanced order, two intermittent running bouts (45 min total) in a hot environment (30.0°C WBGT) in IC and SC. Thereafter, participants sat for 40 min in the hot ambient environment. Repeated measures ANOVA revealed significantly greater micro-environment temperature (p = 0.02) (IC 33.3 ± 3.2°C, SC 32.0 ± 2.8°C) and humidity (p = 0.04) (IC 48.4 ± 8.1%, SC 42.9 ± 7.9%) in IC during the exercise trial but no difference in the 40-min recovery period for micro-environment temperature (p = 0.25) or humidity (p = 0.18). No significant difference (p > 0.05) was shown for core temperature (T (rec)) (IC 38.3 ± 0.4°C, SC 38.2 ± 0.4°C), HR (IC l54 ± 28 beats min(-1), SC 151 ± 26 beats min(-1)) or RPE (IC 4.7 ± 2.1, SC 3.8 ± 1.7) during the exercise trial or recovery period. Results from a paired t test revealed a significantly greater (p < 0.05) S-RPE (IC 5.8 ± 1.2, SC 4.3 ± 1.9), sweat loss (IC 1.4 ± 0.4 L h(-1), SC 1.2 ± 0.4 L h(-1)) and greater discomfort during the exercise and recovery period for the IC. IC clothing appears to have no detrimental effects on heat storage or heat strain during exercise or recovery.

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