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to furnish conditions under which the patient has the best chance to recover. That is the main object of this method. The series of experiments with regard to bone repair that have been reported may contribute considerably to knowledge of the details of repair. The method devised by me in 1921-1922 and first reported in 1923 consists not only of infrequent dressings with aseptic (not antiseptic) petrolatum gauze packs but of an adequate drainage operation when required, immobilization to prevent trauma during and following operation and prolonged protection of the patient against irritative motion, muscle spasm, secondary wound infection and deformity. It is the combination of these factors and not the use of any one or two that con¬ stitutes the Orr method.That retinitis occurs with some forms of toxemia of pregnancy is recognized. However, the clinical significance of such retinitis is not well understood. There have been two main sources of difficulty in the interpretation of its relation to the underlying disease: (1) the varying terminology used by ophthalmologists to describe and designate the retinitis, and (2) the varying classifications of toxemia by obstetricians and clinicians. Although greater uniformity is being gradually attained in both of these phases, it is still hard to avoid the rather meaningless term albuminuric retinitis, and it is still difficult immediately to classify individual cases of toxemia with absolute accuracy. It does not seem feasible at the present time to attempt to prove that retinal lesions occur exclusively in any one type of toxemia of pregnancy. It does seem, however, that the changes seen in the retinas of patients with toxemia are sufficiently constant to furnish, if properly interpreted from the clinical standpoint, valuable aid in the estimation of the patient's immedi¬ ate and future course. I believe that clinical interpre-Fig. 1.-Residual phase of retinitis of toxemia of pregnancy. Marked spas¬ tic and sclerotic narrow¬ ing of the arterioles is shown. Diffuse edema of the retina is still present. tation should be based primarily on the arterioles of the retina, and only secondarily on the retinitis.Miller,1 in 1915,stated: "It has been my experience that albu¬ minurie retinitis of pregnancy affords evidence strongly indica¬ tive of primary nephritis, al¬ though it is not always present in cases of nephritic toxemia." Seemingly, he properly includes in the term albuminurie retinitis scattered hemorrhagic and exu¬ dative areas in the retina as well as the completely developed picture of the retinitis. Cheney,2 in 1924, stated that if a patient with toxemia has retinitis, the chances are four to one that she also has nephritis. He agreed with Schiötz,3 however, that retinitis does occur in cases of acute toxemia of preg¬ nancy without evidence of preexisting nephritis, and From the Section on Ophthalmology, the Mayo Clinic.

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