Neurologic aspects of temporal arteritis

Hollenhorst, Robert W.; Brown, Joe R.; Wagener, Henry P.; Shick, Richard M.

doi:10.1212/wnl.10.5.490

Cited by 193 publications

(65 citation statements)

References 0 publications

Supporting

Mentioning

Contrasting

Unclassified

Order By: Relevance

“…Ptosis in polymyalgia rheumatica and giant cell arteritis is rare. Few cases were described by Hollenhorst et al (4). In their patients ptosis occurred concomitantly with weakness of extraocular muscles supplied by the oculomotor nerve.…”

Section: Giant Cell Arteritis In a Black Personmentioning

confidence: 94%

Giant Cell Arteritis in a Black Person

Dimant

Farmer

Sobol

1978

Arthritis & Rheumatism

View full text Add to dashboard Cite

39 I testing showed a very high degree of concordance, 28.7, 20.4, and 19.5, respectively. The results obtained by the HAM correlated poorly with the other three tests (Figure 2); any correlation of HAM with any other method was substantially poorer than the three correlations described above.We did this study to compare common clinically used tests, in routine daily use, with the simple inexpensive CL test. The findings indicate that the CL method, the FA, and the MFM gave comparable results. Although the CL method appears less sensitive than the MFM and the FA, it seems likely that different populations of antibodies are being measured by the latter methods, particularly antibodies to determinants associated with single-stranded DNA. The HAM, as here employed, correlated poorly with the other techniques and probably is too unreliable for clinical purposes.This study supports the suitability of the CL method for the routine clinical detection of antibodies to double stranded DNA.Our indebtedness to Dr. James Knoke for helpful discussions is acknowledged. WILLIAM CROWE IRVING KUSHNER Giant Cell Arteritis in a Black PersonTo the Editor: To date, giant cell arteritis has been reported only to whites (1,2). A literatures search has failed to show any cases reported in blacks. We wish to report a case of giant cell arteritis in a black person.A 70-year-old black woman, born in Jamaica, West Indies, was admitted to Kings County Hospital because of sudden pain and blurring of vision in the left eye of one day duration. Two months before admission she developed throbbing headaches. Initially the headaches were intermittent and localized to the right side of the head. Later they became constant and diffuse and were partially relieved by aspirin. One month prior to admission, tender, hard lumps developed over the right temporal area, and she had severe pain on mastication. Blurred vision and ptosis occurred the day before admission. Relevant history included hypertension for 18 years, which had been treated with diuretics and several other medications, and nasal obstruction with sinusitis which responded to norepinephrine for 6 years. Family history revealed that the patient's sister had systemic lupus erythematosus.

show abstract

Section: Giant Cell Arteritis In a Black Personmentioning

confidence: 94%

Giant Cell Arteritis in a Black Person

Dimant

Farmer

Sobol

1978

Arthritis & Rheumatism

View full text Add to dashboard Cite

show abstract

“…Em 1960, Hollenhorst e col. 4 , estudando 175 exemplares com arterite craniana, descreveram dois com paralisias de membros superiores, interrogando se estas seriam neuríticas ou miosíticas. Warren e col. lc », em 1968, registraram dois casos com lesões do sistema nervoso periférico: o primeiro com mononeuropatia e o segundo com polineuropatia.…”

Section: Comentáriosunclassified

Neuropatia múltipla por arterite de células gigantes: registro clínico-patológico de um caso

Filho

Freitas

Caldas

et al. 1978

Arq. Neuro-Psiquiatr.

View full text Add to dashboard Cite

A arterite de células gigantes, também conhecida como arterite temporal ou arterite craniana, é afecção possivelmente de natureza auto-imune, de caráter inflamatório, ocorrendo geralmente em pacientes acima dos 55 anos de idade. Clinicamente traduz-se por quadro de hipertermia discreta, anorexia, perda de peso, dor no trajeto da artéria temporal superficial uni ou bilateralmente e, com menor freqüência, dor ao nível da artéria occipital. Estas artérias encontram-se quase sempre tortuosas, dolorosas à palpação e endurecidas pela presença de nódulos em seus trajetos. A velocidade de hemossedimentação está geralmente elevada, ocorrendo ainda leucocitose, discreta anemia hipocrômica e, em alguns casos, aumento da alfa-globulina sérica. O diagnóstico é confirmado pela biópsia da artéria temporal superficial que mostra espessamento da íntima com presença de infiltrado inflamatório de células gigantes na intima e na camada média, necrose focal com fragmentação e duplicação da lâmina elástica interna. As artérias nutrientes dos nervos ópticos são acometidas em 30% dos casos provocando perda de visão uni ou bilateralmente.A primeira descrição da afecção em apreço deve-se a Hutchinson« em 1890. Entretanto foram Horton, Magath e Brown 6 em 1934 que a individualizaram como entidade clinica. Sabe-se hoje que tal moléstia não está restrita às artérias do sistema carotídeo. Outros vasos arteriais de médio e grosso calibres como a aorta, artérias vertebrais, coronarianas, hepáticas e renais podem ser acometidas. É, assim, a arterite de células gigantes, uma afecção de comprometimento do sistema vascular «,4,8,9,10, Precedendo ou acompanhando o cortejo sintomático, alguns pacientes queixam-se de dor muscular, principalmente localizada na cintura escapular, quadro álgico este denominado pelos reumatologistas polimialgia reumática. O uso precoce de pequenas doses de corticosteróides contribui para a melhora deste polimorfismo sintomático, tornando mais raras as graves complicações da afecção em apreço. A observação de paciente de 67 anos de idade com o diagnóstico clínico-patológico da arterite craniana, que

show abstract

“…Temporal artery involvement, with headaches and blindness resulting from involvement of the posterior ciliary and ophthalmic arteries, is a frequent finding (1)(2)(3)(4)(5)(6)(7). A feature of GCA that is not widely recognized is that it affects the large arteries diffusely and may be the cause of claudication, aneurysmal dilatation, and aortic aneurysm dissection and rupture (2,(8)(9)(10)(11)(12)(13)(14).…”

mentioning

confidence: 99%