The purpose of this study was to examine the effect of environmental temperature on variability in power output, self-selected pacing strategies, and performance during a prolonged cycling time trial. Nine trained male cyclists randomly completed four 40 km cycling time trials in an environmental chamber at 17°C, 22°C, 27°C, and 32°C (40% RH). During the time trials, heart rate, core body temperature, and power output were recorded. The variability in power output was assessed with the use of exposure variation analysis. Mean 40 km power output was significantly lower during 32°C (309 ± 35 W) compared with 17°C (329 ± 31 W), 22°C (324 ± 34 W), and 27°C (322 ± 32 W). In addition, greater variability in power production was observed at 32°C compared with 17°C, as evidenced by a lower (P = .03) standard deviation of the exposure variation matrix (2.9 ± 0.5 vs 3.5 ± 0.4 units, respectively). Core temperature was greater (P < .05) at 32°C compared with 17°C and 22°C from 30 to 40 km, and the rate of rise in core temperature throughout the 40 km time trial was greater (P < .05) at 32°C (0.06 ± 0.04°C·km–1) compared with 17°C (0.05 ± 0.05°C·km–1). This study showed that time-trial performance is reduced under hot environmental conditions, and is associated with a shift in the composition of power output. These finding provide insight into the control of pacing strategies during exercise in the heat.
This investigation describes the sprint performances of the highest internationally ranked professional male road sprint cyclist during the 2008–2011 Grand Tours. Sprint stages were classified as won, lost, or dropped from the front bunch before the sprint. Thirty-one stages were video-analyzed for average speed of the last km, sprint duration, position in the bunch, and number of teammates at 60, 30, and 15 s remaining. Race distance, total elevation gain (TEG), and average speed of 45 stages were determined. Head-to-head performances against the 2nd–5th most successful professional sprint cyclists were also reviewed. In the 52 Grand Tour sprint stages the subject started, he won 30 (58%), lost 15 (29%), was dropped in 6 (12%), and had 1 crash. Position in the bunch was closer to the front and the number of team members was significantly higher in won than in lost at 60, 30, and 15 s remaining (P < .05). The sprint duration was not different between won and lost (11.3 ± 1.7 and 10.4 ± 3.2 s). TEG was significantly higher in dropped (1089 ± 465 m) than in won and lost (574 ± 394 and 601 ± 423 m, P < .05). The ability to finish the race with the front bunch was lower (77%) than that of other successful sprinters (89%). However, the subject was highly successful, winning over 60% of contested stages, while his competitors won less than 15%. This investigation explores methodology that can be used to describe important aspects of road sprint cycling and supports the concept that tactical aspects of sprinting can relate to performance outcomes.
Purpose:Heat stress might attenuate the effects of carbohydrate on immunoendocrine responses to exercise by increasing endogenous glucose production and reducing the rate of exogenous carbohydrate oxidation. The authors compared the efficacy of carbohydrate consumption on immune responses to exercise in temperate vs. hot conditions.Methods:Ten male cyclists exercised on 2 separate occasions in temperate (18.1 ± 0.4 °C, 58% ± 8% relative humidity) and on another 2 occasions in hot conditions (32.2 ± 0.7 °C, 55% ± 2% relative humidity). On each occasion, the cyclists exercised in a fed state for 90 min at ~60% VO2max and then completed a 16.1-km time trial. Every 15 min during the first 90 min of exercise, they consumed 0.24 g/kg body mass of a carbohydrate or placebo gel.Results:Neutrophil counts increased during exercise in all trials (p < .05) and were significantly lower (40%, p = .006) after the carbohydrate than after the placebo trial in 32 °C. The concentrations of serum interleukin (IL)-6, IL-8, and IL-10 and plasma granulocyte-colony-stimulating factor, myeloperoxidase, and calprotectin also increased during exercise in all trials but did not differ significantly between the carbohydrate and placebo trials. Plasma norepinephrine concentration increased during exercise in all trials and was significantly higher (50%, p = .01) after the carbohydrate vs. the placebo trial in 32 °C.Conclusion:Carbohydrate ingestion attenuated neutrophil counts during exercise in hot conditions, whereas it had no effect on any other immune variables in either temperate or hot conditions.
A number of laboratory-based performance tests have been designed to mimic the dynamic and stochastic nature of road cycling. However, the distribution of power output and thus physical demands of high-intensity surges performed to establish a breakaway during actual competitive road cycling are unclear. Review of data from professional road-cycling events has indicated that numerous short-duration (5–15 s), high-intensity (~9.5–14 W/kg) surges are typically observed in the 5–10 min before athletes’ establishing a breakaway (ie, riding away from a group of cyclists). After this initial high-intensity effort, power output declined but remained high (~450–500 W) for a further 30 s to 5 min, depending on race dynamics (ie, the response of the chase group). Due to the significant influence competitors have on pacing strategies, it is difficult for laboratory-based performance tests to precisely replicate this aspect of mass-start competitive road cycling. Further research examining the distribution of power output during competitive road racing is needed to refine laboratory-based simulated stochastic performance trials and better understand the factors important to the success of a breakaway.
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