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In 16 patients with abdominal trauma, ultrasonic abnormalities of the spleen were demonstrated shortly after trauma (mean 3.7 days). Initial sonographic abnormalities included: splenic laceration in 3 cases, intrasplenic fluid (hematoma) in 4, splenic inhomogeneity (contusion) in 8, perisplenic fluid (subcapsular hematoma) in 11, intraperitoneal fluid in 10, and a left pleural effusion in 7. Follow-up sonograms showed that pleural effusions and intraperitoneal fluid collections disappeared quickly (2 and 4 weeks, respectively). However, intrasplenic hematomas and contusions usually resorbed over a period of months (up to a year). When followed to complete resolution, the spleen may become normal sonographically or there may be small linear foci of echogenic material, which probably represent scar tissue.

The SAGE libraries reported in this article provide the gene expression profile of NHMCs and HMLCs. It thus represents a valuable source of information regarding the function of Müller cells as well as their role in the development of diabetic retinopathy.

HMCLs can be used as a model to improve the understanding of Müller cells in the context of chronic diabetes.

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