Tuberculous pericarditis∗

Schepers, G. W. H.

doi:10.1016/0002-9149(62)90044-9

Cited by 59 publications

(13 citation statements)

References 87 publications

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“…40 Earlier studies suggested that tuberculous pericarditis was more readily diagnosed from pericardial biopsy specimens than from fluid alone. 41 A prospective comparison of pericardial fluid culture and histology in a TB endemic area has demonstrated, however, that culture of the fluid confirmed TB more often than did histology of the pericardium. 31 Corey et al 28 have demonstrated that tissue histology was more important primarily in cases in which no pericardial fluid could be obtained.…”

Section: Direct Methods For the Diagnosis Of A Tuberculous Pathogenesismentioning

confidence: 99%

“…37,42 The diagnostic sensitivity for TB by pericardial biopsy ranges from 10% to 64%. 29,41,43 Therefore, a normal pericardial biopsy specimen does not exclude tuberculous pericarditis because in some patients, examination of the pericardium removed at pericardiectomy or autopsy is required to demonstrate clear-cut evidence of TB. 5,44 The polymerase chain reaction (PCR) has also been suggested for detecting M tuberculosis DNA in pericardial fluid.…”

Section: Direct Methods For the Diagnosis Of A Tuberculous Pathogenesismentioning

confidence: 99%

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Tuberculous Pericarditis

2005

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Background-The incidence of tuberculous pericarditis is increasing in Africa as a result of the human immunodeficiency virus (HIV) epidemic. The primary objective of this article was to review and summarize the literature on the pathogenesis, diagnosis, and management of tuberculous pericarditis. Methods and Results-We searched MEDLINE (January 1966 to May 2005 and the Cochrane Library (Issue 1, 2005) for information on relevant references. A "definite" diagnosis of tuberculous pericarditis is based on the demonstration of tubercle bacilli in pericardial fluid or on a histological section of the pericardium; "probable" tuberculous pericarditis is based on the proof of tuberculosis elsewhere in a patient with otherwise unexplained pericarditis, a lymphocytic pericardial exudate with elevated adenosine deaminase levels, and/or appropriate response to a trial of antituberculosis chemotherapy. Treatment consists of the standard 4-drug antituberculosis regimen for 6 months. It is uncertain whether adjunctive corticosteroids are effective in reducing mortality or progression to constriction. Surgical resection of the pericardium remains the appropriate treatment for constrictive pericarditis. The timing of surgical intervention is controversial, but many experts recommend a trial of medical therapy for noncalcific pericardial constriction, and pericardiectomy in nonresponders after 4 to 8 weeks of antituberculosis chemotherapy.

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Section: Direct Methods For the Diagnosis Of A Tuberculous Pathogenesismentioning

confidence: 99%

Section: Direct Methods For the Diagnosis Of A Tuberculous Pathogenesismentioning

confidence: 99%

Tuberculous Pericarditis

2005

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“…Anatomopathology of the pericardial biopsy specimen make certitude diagnosis of TB, but thoracotomy and all its complications with increased morbidity and marked prolongation of the hospital stay may limit the procedure [16]. However the sensitivity diagnostic of pericardial biopsy for TP ranges only from 10% to 64% [17,18]. In other more valuable diagnosis tool, we find the polymerase chain reaction (PCR) for detecting Mycobacterium tuberculosis DNA in pericardial effusion fluid [19].…”

Section: Discussionmentioning

confidence: 99%

Tuberculous pericarditis: An exceptional STEMI association: Case report

Sanoussi¹,

Kourireche²,

Lakhal³

et al. 2017

Interventional-Cardiology

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Among all the complications of myocardial infarction, pericardial effusion remains correlated with a high morbidity and mortality rate. Tuberculosis in Morocco is a public health problem with 25,000 new cases per year, 47% of which are extra pulmonary, we present an exceptional case of a 70 year old man presenting with an extensive anterior myocardial infarction complicated with tuberculous pericarditis effusion, highlighting the importance of thinking of this aetiology in a specific context.

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“…In general, tuberculous lesions erode into blood vessels, disseminating millions of tubercle bacilli into the bloodstream (7) and the pericardium, as in this case. Moreover, up to 90% of patients with tuberculous pericarditis who are left untreated die within the first year due to dissemination of M. tuberculosis or the development of congestive heart failure resulting from constrictive pericarditis (11).…”

Section: Discussionmentioning

confidence: 99%

Sudden Cardiac Arrest Caused by Tuberculous Pericarditis with Hemorrhagic Pericardial Effusion

et al. 2012

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As tuberculosis still exists in Japan, tuberculous pericarditis is a major health issue. Tuberculous pericarditis is difficult to diagnose and leads to poor outcomes when left untreated. We herein report the case of a patient who was admitted to the hospital after undergoing resuscitation for cardiopulmonary arrest. Mycobacterium tuberculosis was detected in his hemorrhagic pericardial fluid and tuberculous pericarditis was diagnosed. The administration of antituberculous medication resulted in marked improvements. A diagnosis of tuberculous pericarditis, in addition to other causes such as malignant tumors, should therefore be considered in the differential diagnosis for cases presenting with hemorrhagic pericardial effusion, even in those involving sudden cardiac arrest.

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Tuberculous pericarditis∗

Cited by 59 publications

References 87 publications

Tuberculous Pericarditis

Tuberculous Pericarditis

Tuberculous pericarditis: An exceptional STEMI association: Case report

Sudden Cardiac Arrest Caused by Tuberculous Pericarditis with Hemorrhagic Pericardial Effusion

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