The renal excretory activity of atrial natriuretic factor is independent of renal prostaglandins in humans

Castellani, Sergio; Rosso, Angela Del; Chen, J.L.; Camaiti, Alberto; Carnovali, M; Masotti, Giulio

doi:10.1016/0952-3278(91)90062-a

Prostaglandins, Leukotrienes and Essential Fatty Acids

1991

DOI: 10.1016/0952-3278(91)90062-a

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The renal excretory activity of atrial natriuretic factor is independent of renal prostaglandins in humans

Sergio Castellani¹,

Angela Del Rosso²,

J.L. Chen³

et al.

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1992

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Cited by 5 publications

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“…Immediately after collection, the urine was frozen and stored at Ϫ20°C until extraction and purification procedures could be carried out. Renal thromboxane B 2 (TxB 2 ) was measured by radioimmunoassay (RIA), as reported elsewhere (27). A tritium tracer RIA was performed after allowing four to five months for the radioactivity of the iodine-125 and -131 in the urine to decay to background levels so as not to interfere with the TxB 2 RIA (28).…”

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confidence: 99%

Thromboxane inhibition improves renal perfusion and excretory function in severe congestive heart failure

Castellani¹,

Paniccia²,

Serio³

et al. 2003

Journal of the American College of Cardiology

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confidence: 99%

Thromboxane inhibition improves renal perfusion and excretory function in severe congestive heart failure

Castellani¹,

Paniccia²,

Serio³

et al. 2003

Journal of the American College of Cardiology

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Neuroendocrine response to diclofenac in healthy subjects: a pilot study

Varaldo

Sibilla

Bioletto

et al. 2023

J Endocrinol Invest

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Purpose The precise effects of non-steroidal anti-inflammatory drugs on the neuroendocrine hydro-electrolytic regulation are not precisely understood. The aim of this pilot study was to evaluate, in healthy subjects, the neuroendocrine response of the antidiuretic system to intravenous diclofenac infusion. Methods For this single-blinded, cross-over study, we recruited 12 healthy subjects (50% women). Test sessions were divided into three observation times (pre-test; test; 48 h post-test), which were repeated equally on two different occasions, with the administration of diclofenac (75 mg in saline solution 0.9% 100 cc) on 1 day, or placebo (saline solution 0.9% 100 cc) on another day. The night before the test the subjects were asked to collect a salivary cortisol and cortisone sample, which was repeated on the night of the procedure session. Serial urine and blood samples were collected on the test day (for osmolality, electrolytes, ACTH, cortisol, copeptin, MR-proADM, MR-proANP; the last three represent more stable and analytically reliable molecules than their respective active peptides). Moreover, the subjects were evaluated with the bioimpedance vector analysis (BIVA) before and after the test. Forty-eight hours after the end of the procedure urine sodium, urine potassium, urine osmolality, serum sodium and copeptin were revaluated together with BIVA. Results No significant changes in circulating hormone levels were observed; anyway, 48 h after diclofenac, BIVA showed a significant water retention (p < 0.00001), especially in extracellular fluid (ECF) (16.47 ± 1.65 vs 15.67 ± 1.84, p < 0.001). Salivary cortisol and cortisone tended to increase only the night after placebo administration (p = 0.054 cortisol; p = 0.021 cortisone). Conclusion Diclofenac resulted in an increased ECF at 48 h, but this phenomenon seems to be associated with a greater renal sensibility to the action of vasopressin rather than with an increase in its secretion. Moreover, a partial inhibitory effect on cortisol secretion can be hypothesized.

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Increased renal formation of thromboxane A 2 and prostaglandin F 2α in heart failure

Castellani¹,

Paladini²,

Paniccia³

et al. 1997

American Heart Journal

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The renal excretory activity of atrial natriuretic factor is independent of renal prostaglandins in humans

Cited by 5 publications

References 28 publications

Thromboxane inhibition improves renal perfusion and excretory function in severe congestive heart failure

Thromboxane inhibition improves renal perfusion and excretory function in severe congestive heart failure

Neuroendocrine response to diclofenac in healthy subjects: a pilot study

Increased renal formation of thromboxane A 2 and prostaglandin F 2α in heart failure

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