Saturation of human saliva with respect to calcium salts

Larsen, Mona; Pearce, E.I.F.

doi:10.1016/s0003-9969(03)00007-4

Cited by 118 publications

(94 citation statements)

References 38 publications

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“…In the present study the pH of the bleaching agent was around 6.5. Considering that the critical pH for enamel demineralization is around 5.5, 23 and the pH of the agent in this study was higher than the critical pH, it is probable that this fact has influenced the lack of changes in microhardness in the present study. In a study in which the effect of Opalescence bleaching agent with a pH value of 6.5 was compared with that of Rembrandt having a pH value of 4.9 on enamel microhardness, Opalescence increased and Rembrandt decreased microhardness.…”

Section: Discussionmentioning

confidence: 74%

In Vitro Evaluation of the Effect of Delaying Toothbrushing With Toothpaste on Enamel Microhardness Subsequent to Bleaching the Teeth With 15% Carbamide Peroxide

Navimipour

Kimyai²,

Nikazar³

et al. 2012

Operative Dentistry

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Delaying oral hygiene procedures during bleaching does not seem to cause any change in enamel microhardness. SUMMARYChanges in enamel surface microhardness as a result of bleaching with carbamide peroxide in various in vitro conditions have been reported. The present study evaluated the effect of oral hygiene procedures on enamel microhardness at three time intervals following bleaching with 15% carbamide peroxide. Although this was an in vitro study, the purpose was to address whether or not a patient's toothbrushing following at-home bleaching might affect surface changes in tooth enamel. Eighty enamel slabs were prepared from impacted human third molars that had been extracted surgically.

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Section: Discussionmentioning

confidence: 74%

In Vitro Evaluation of the Effect of Delaying Toothbrushing With Toothpaste on Enamel Microhardness Subsequent to Bleaching the Teeth With 15% Carbamide Peroxide

Navimipour

Kimyai²,

Nikazar³

et al. 2012

Operative Dentistry

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show abstract

“…When 10 volunteers imbibed 1 l of a soft acidic drink over a period of 15 days, some volunteers showed minimal erosion of enamel and others far more erosion [156]. Identification of the cause of susceptibility is unknown but likely to be due to salivary buffering and flow rate [157], pellicle thickness and charge [158], movements of the soft tissues [159], distribution and time of acidic liquid in the oral cavity [160], tooth structure [139] and remineralising potential [161, 162]. Difference in saliva flow and composition could contribute to the development of hypersensitivity by affecting the surface layer or deposition of intratubular dentin [163].…”

Section: Susceptibility and Risk Factorsmentioning

confidence: 99%

Dentin hypersensitivity: pain mechanisms and aetiology of exposed cervical dentin

et al. 2012

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ObjectivesThe paper’s aim is to review dentin hypersensitivity (DHS), discussing pain mechanisms and aetiology.Materials and methodsLiterature was reviewed using search engines with MESH terms, DH pain mechanisms and aetiology (including abrasion, erosion and periodontal disease).ResultsThe many hypotheses proposed for DHS attest to our lack of knowledge in understanding neurophysiologic mechanisms, the most widely accepted being the hydrodynamic theory. Dentin tubules must be patent from the oral environment to the pulp. Dentin exposure, usually at the cervical margin, is due to a variety of processes involving gingival recession or loss of enamel, predisposing factors being periodontal disease and treatment, limited alveolar bone, thin biotype, erosion and abrasion.ConclusionsThe current pain mechanism of DHS is thought to be the hydrodynamic theory. The initiation and progression of DHS are influenced by characteristics of the teeth and periodontium as well as the oral environment and external influences. Risk factors are numerous often acting synergistically and always influenced by individual susceptibility.Clinical relevanceWhilst the pain mechanism of DHS is not well understood, clinicians need to be mindful of the aetiology and risk factors in order to manage patients’ pain and expectations and prevent further dentin exposure with subsequent sensitivity.

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“…Calcium ions are among the components of the pellicle layer; they are present in human saliva and are chelated by salivary proteins. 47 Furthermore, it is known that the pellicle layer can be partially removed from the tooth surface during acid-induced demineralization, 48 thus the components of the pellicle layer appear in the acidic solutions. The analysis of calcium concentrations in the acidic solution provides the total calcium content, where some of the calcium ions were released by the demineralized enamel tissue, but some of them could have originated from the detached salivary pellicle layer.…”

Section: Optical Erosion Analysis In Enamel Samplesmentioning

confidence: 99%

Effects of enamel abrasion, salivary pellicle, and measurement angle on the optical assessment of dental erosion

et al. 2012

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Abstract. The present study assessed the effects of abrasion, salivary proteins, and measurement angle on the quantification of early dental erosion by the analysis of reflection intensities from enamel. Enamel from 184 caries-free human molars was used for in vitro erosion in citric acid (pH 3.6). Abrasion of the eroded enamel resulted in a 6% to 14% increase in the specular reflection intensity compared to only eroded enamel, and the reflection increase depended on the erosion degree. Nevertheless, monitoring of early erosion by reflection analysis was possible even in the abraded eroded teeth. The presence of the salivary pellicle induced up to 22% higher reflection intensities due to the smoothing of the eroded enamel by the adhered proteins. However, this measurement artifact could be significantly minimized (p < 0.05) by removing the pellicle layer with 3% NaOCl solution. Change of the measurement angles from 45 to 60 deg did not improve the sensitivity of the analysis at late erosion stages. The applicability of the method for monitoring the remineralization of eroded enamel remained unclear in a demineralization/remineralization cycling model of early dental erosion in vitro.

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Saturation of human saliva with respect to calcium salts

Cited by 118 publications

References 38 publications

In Vitro Evaluation of the Effect of Delaying Toothbrushing With Toothpaste on Enamel Microhardness Subsequent to Bleaching the Teeth With 15% Carbamide Peroxide

In Vitro Evaluation of the Effect of Delaying Toothbrushing With Toothpaste on Enamel Microhardness Subsequent to Bleaching the Teeth With 15% Carbamide Peroxide

Dentin hypersensitivity: pain mechanisms and aetiology of exposed cervical dentin

Effects of enamel abrasion, salivary pellicle, and measurement angle on the optical assessment of dental erosion

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