Physiology of Pain

Sembulingam, K

doi:10.5005/jp/books/10283_145

Cited by 7 publications

(10 citation statements)

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“…On the other hand, when testosterone production is low, lack of inhibition of hypothalamus leads to secretion of testosterone through LHRH and LH. 28 2/58 (3.44%) of the azoospermic men had low serum gonadotrophins (LH & FSH) and hyperprolactinamia but low testosterone. This was closely related to the findings of the studies of Geidam A.D et al in 2008 where a prevalence of 4/58 (4.2%) patients having hypogonadotrophic hypogonadism among their study group.…”

Section: (F) Studies By Masanori Et Al In 1995 Revealed That Azoospementioning

confidence: 99%

“…23 Hypogonadotropic hypogonadism is a result of decreased gonadotropin stimulation of potentially normal testes. 28,29 It can be caused by hypothalamic disorders, e.g., congenital abnormalities such as Kallmann syndrome or acquired pituitary disorders, e.g., functioning and non-functioning pituitary tumors. 9 As shown in our result (Table 1D), hypogonadism and normogonadotropin associated with high prolactin are not a very common profile pattern but are of clinical interest.…”

Section: (F) Studies By Masanori Et Al In 1995 Revealed That Azoospementioning

confidence: 99%

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The Prevalence and Patterns of Endocrinopathies Amongs Azoospermic Male Partners at a Fertility Clinic in Benin City

Uhuonrenren¹

2014

EMIJ

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Aim and Objective: We undertook this study to evaluate the prevalence, trend and patterns of endocrine abnormalities among azoospermic male partners of infertile couples investigated for male infertility in our fertility centre. Setting: A private IVF hospital treating infertility with assisted reproduction technology in Benin City, Nigeria. Methodology: A prospective, observational and a descriptive study of the analysis of the hormonal profile of azoospermic male partners of the infertile couples, who presented at St Augustine Medical center, Benin City, Nigeria over a five-year period from May 2007 through December 2012 was undertaken. Results: A total of 686 men were investigated for infertility within this period, fifty-eight (8.45%) of them with a mean age of 43.72±1.5years were found to be azoospermic. Of the 58 azoospermic men, fourty-eight (82.76%) had one or more abnormal hormonal profile parameter, while ten (17.24%) had a normal hormone profile pattern. Of the forty-eight Azoospermic men with abnormal hormone profile pattern, twenty-two (37.93%) had increased serum FSH and LH, mildly increased prolactin and low testosterone, 15 (25.86%) had increased serum FSH, normal LH and prolactin with reduced testosterone, 4 (6.9%) had increased serum LH, normal FSH and prolactin with reduced testosterone, 2 (3.45%) had low LH, FSH and testosterone with high prolactin while 5 (10.42%) had elevated prolactin levels, normal gonadotrophins (LH & FSH) and low testosterone. Ejaculate volume was normal in 48(82.76%) of the azoospermic men when the mean semen volume was 3.30±0.19mls; and mean serum hormone levels were LH; 9.54 ± 0.89, FSH; 22.77± 2.70, Prolactin; 18.54± 2.50, Testosterone; 3.01 ± 0.22. The ejaculate volume was low in 10 (17.24%) men with a mean semen volume of 0.89±0.07 and mean serum hormone levels;

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Section: (F) Studies By Masanori Et Al In 1995 Revealed That Azoospementioning

confidence: 99%

Section: (F) Studies By Masanori Et Al In 1995 Revealed That Azoospementioning

confidence: 99%

The Prevalence and Patterns of Endocrinopathies Amongs Azoospermic Male Partners at a Fertility Clinic in Benin City

Uhuonrenren¹

2014

EMIJ

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“…Certain number of neurons in hypothalamus are believed to be sensitive to testosterone and estrogen levels. 13 The yield of sperms is controlled by luteinizing hormone that is formed in pituitary gland. Its mechanism of action is that it acts on leydig cells to produce testosterone which binds to sertoli cells and stimulates production of sperms.…”

Section: Doimentioning

confidence: 99%

Semen Parameters;

Sultan¹,

Khan²,

Ahmed³

2018

TPMJ

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Method: A total of 70 infertile men were included in this study. Semen samples for semen analysis and serum for hormonal analysis were obtained and further tests were performed. Results: The results of our study show that 28.57% were Normospermic, 18.56% were azoospermic and 52.85% were Oligospermic. In Oligospermic and azoospermic patients significant low levels of testosterone and prolactin were noticed. The results indicate that along with semen parameters hormonal levels should also be assessed to diagnose the underlying cause of infertility. Conclusion: It is concluded from our study that both quality of semen and hormonal levels cause infertility in men. Therefore the patients should be properly investigated and should be treated accordingly to the underlying abnormality. This is a regional study and further studies should be conducted to have a conclusive evidence at national level.

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“…Hypothalamic stimulation may induce receptivity in females and simple copulatory movements in males. Some hypothalamic neurons are sensitive to circulating oestrogen or testosterone [13].…”

Section: Introductionmentioning

confidence: 99%

“…When LHRH secretion is inhibited, LH is not released from anterior pituitary, resulting in the stoppage of testosterone secretion from testes. On the other hand, when testosterone production is low, lack of inhibition of hypothalamus leads to secretion of testosterone through LHRH and LH [13].…”

Section: Introductionmentioning

confidence: 99%

Semen Parameters and Hormaone Profile of Men Investigated for Infertility at Midland Fertility Centre, Ilorin, Nigeria

Omotoso¹

2012

J. Basic Appl. Sci.

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This study aimed at comparing the semen parameters and pattern of endocrine abnormalities in patients investigated for male infertility in our fertility centre. Hormonal assays and semen analysis were carried out, from February 2008 to April 2010. Three hundred and sixteen (316) men were evaluated out of which forty-five (45) underwent hormonal assessment, because of the abnormalities in their sperm count. Sixteen (35.6%; 16 out of 45) of the subjects were azoospermic; twenty-three (23 out of 45; 51.1%) were oligospermic with sperm count less than 20 million sperm cell/ ml; and six (n=6; 13.3%) of the subjects were normospermic, with sperm count greater than 20 million sperm cell/ ml. The mean values of the hormonal assays for azoospermia were: 21.84±1.94 (luteinizing hormone), 14.14±4.4 (follicle-stimulating hormone), 23.95±17.43 (testosterone), and 15.03±2.91 for prolactin. Hormonal assays for patients having oligospermia were as follow: luteinizing hormone (LH) 12.56±3.90, follicle-stimulating hormone (FSH) 5.96±1.17, testosterone 10.13±2.40 and prolactin 13.42±2.43, while normospermic patients had the following hormone levels: LH: 7.72±3.90, FSH: 2.93±.74, testosterone: 17.00±3.36 and, prolactin: 12.45±2.16. We conclude that infertile men with low sperm count may not always present with abnormal hormone profiles; and, male factor, in this environment, contributes a high percentage to infertility.

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Physiology of Pain

Cited by 7 publications

References 0 publications

The Prevalence and Patterns of Endocrinopathies Amongs Azoospermic Male Partners at a Fertility Clinic in Benin City

The Prevalence and Patterns of Endocrinopathies Amongs Azoospermic Male Partners at a Fertility Clinic in Benin City

Semen Parameters;

Semen Parameters and Hormaone Profile of Men Investigated for Infertility at Midland Fertility Centre, Ilorin, Nigeria

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