Ovarian Hyperstimulation Syndrome (OHSS)

Abstract

Précis1. Clinical Setting: abdominal bloating and distention, intravascular volume depletion, ascites, hemoconcentration, and oliguria following a fertility intervention involving the administration of exogenous FSH. of a GnRH agonist to stimulate endogenous LH secretion to trigger ovarian follicle luteinization in order to reduce ovarian production of VEGF. Cessation of FSH stimulation (coasting), in order to reduce the ovarian production of VEGF. Freezing all embryos and cancelling embryo transfer to avoid a late case of OHSS caused by pregnancy. Administration of a dopamine agonist such as cabergoline to reduce ovarian production of VEGF.

Age‐specific serum anti‐Müllerian hormone concentration in Japanese women and its usefulness as a predictor of the ovarian response

“…It is important to predict and prevent OHSS as the associated morbidity can be severe. Furthermore, improper management can result in severe sequelae . In severe OHSS cases, renal failure leads to decreased urine volume, followed by respiratory and circulatory failure.…”

“…Furthermore, improper management can result in severe sequelae. 8 In severe OHSS cases, renal failure leads to decreased urine volume, followed by respiratory and circulatory failure. Life-threatening thrombosis also can develop with the progression of OHSS.…”