2020
DOI: 10.21203/rs.2.23752/v2
| View full text |Cite
Preprint
|
Sign up to set email alerts
|

Abstract: Background: CaMKIV has been identified as a potential regulator of skeletal muscle glucose metabolism and insulin gene expression in the pancreas. However, the mechanism of CaMKIV involved in adipose insulin resistance is not fully understood. Autophagy has to be shown as a potential therapeutic target for ER (endoplasmic reticulum) stress and insulin resistance. The purpose of this study is to investigate the effects of CaMKIV on the ER stress, autophagic function and insulin signaling in tunicamycin treated … Show more

Help me understand this report
View published versions

Search citation statements

Order By: Relevance

Paper Sections

Select...

Citation Types

0
0
0

Publication Types

Select...

Relationship

0
0

Authors

Journals

citations
Cited by 0 publications
references
References 29 publications
(36 reference statements)
0
0
0
Order By: Relevance

No citations

Set email alert for when this publication receives citations?