Herpes zoster ophthalmicus evolving into headache characterised as hemicrania continua

Prakash, Sanjay; Dave, Ankit; Joshi, Hemant

doi:10.1136/bcr-2018-224235

Cited by 5 publications

(5 citation statements)

References 15 publications

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“…For posttraumatic headache the ICHD defines onset within seven days, but this is not consistent for other secondary pathologies where 'a close temporal relation' remains undefined. Prakash et al (90) reported on two cases of ophthalmic zoster reactivation and simultaneous onset of headache which persisted once the zoster settled. Both cases subsequently responded to indometacin (90).…”

Section: Secondary Hemicrania Continuamentioning

confidence: 99%

“…Prakash et al (90) reported on two cases of ophthalmic zoster reactivation and simultaneous onset of headache which persisted once the zoster settled. Both cases subsequently responded to indometacin (90). In a group of event-related cases of HC, 12 followed head injury, post-stroke, post-partum or a surgical procedure.…”

Section: Secondary Hemicrania Continuamentioning

confidence: 99%

“…Prakash et al. (90) reported on two cases of ophthalmic zoster reactivation and simultaneous onset of headache which persisted once the zoster settled. Both cases subsequently responded to indometacin (90).…”

Section: Hemicrania Continuamentioning

confidence: 99%

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Paroxysmal hemicrania and hemicrania continua: Review on pathophysiology, clinical features and treatment

Bahra

2023

Cephalalgia

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Background Paroxysmal hemicrania and hemicrania continua are indometacin-sensitive trigeminal autonomic cephalalgias, a terminology which reflects the predominant distribution of the pain, observable cranial autonomic features and shared pathophysiology. Understanding the latter is limited, both by low prevalence and the intricacies of studying brain function, requiring multimodal techniques to glean insights into such disorders. Similarly obscure is the curious response to indometacin. This review will address what is currently known about pathophysiology, the rationale for the current classification and, features which may confound the diagnosis, such as lack of cranial autonomic symptoms and those which are typically associated with migraine such as nausea, photophobia, phonophobia and aura. Despite these characteristics, a dramatic response to indometacin, which is not seen in migraine nor the other trigeminal autonomic cephalalgias , provides the hallmark of the diagnosis. The main clinical differential for paroxysmal hemicrania is based on temporal pattern and lies between cluster headache and short-lasting-neuralgiform headache attacks with tearing or additional cranial autonomic symptoms. For hemicrania continua it is more challenging as the main differential for which the disorder is often treated is migraine. A prior episodic pattern, often days at a time, and the tendency to exacerbation with analgesics will further deflect from the diagnosis. The relevance of this is that there is little overlap in therapeutics between paroxysmal hemicrania and hemicrania continua and other headache disorders and there are limited effective alternatives to indometacin. The most effective are other non-steroidal anti-inflammatory drugs including the newer COX-II inhibitors. Even though early reports suggest that a higher indometacin dose-requirement may herald a secondary precipitating pathology, this does not seem to be the case, with syndrome and response to treatment being similar with the primary disorder. In this context imaging of new onset paroxysmal hemicrania or hemicrania continua and implication of the results will be discussed as will alternative treatment options.

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Section: Secondary Hemicrania Continuamentioning

confidence: 99%

Section: Secondary Hemicrania Continuamentioning

confidence: 99%

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Paroxysmal hemicrania and hemicrania continua: Review on pathophysiology, clinical features and treatment

Bahra

2023

Cephalalgia

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“…atvejų [9]. HZ gali būti galvos skausmo (poherpetinės hemicrania continua) priežastis [10]. VZV infekcija sukelia ir atipines manifestacijas − gliomą, zoster nesant bėrimo, abipusę HZ [11].…”

Section: Herpes Ir Influenza Virusinių Infekcijų Neurologinės Komplikacijosunclassified

“…Skirtas indometacinas būklę palengvino. Ligoniams, esant PN ir kranijiniam autonominiam pažeidimui, refrakterinės HZ neuropatijos gydymui rekomenduojama skirti indometaciną [10]. Mūsų pateiktas 78 metų ligonės R.K. klinikinis atvejis patvirtina indometacino veiksmingumą, gydant poherpetinę hemicrania continua.…”

Section: Literatūros Apžvalga Ir Diskusijaunclassified

Herpes Ir Influenza Virusinių Infekcijų Neurologinės Komplikacijos

Kimševaitė¹

2020

Health Sciences

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Straipsnyje analizuojama herpes ir influenza virusinių infekcijų neurologinė patologija. Herpes ir influenza virusinių infekcijų neurologinių komplikacijų spektras platus − nuo neuralgijų iki encefalito, epilepsinės būklės ir komos. Encefalitas yra viena sunkiausių herpes ir influenza virusų sukeliamų komplikacijų. Herpes zoster yra nepriklausomas insulto ir praeinančio smegenų išemijos priepuolio rizikos veiksnys ligoniams iki 40 metų. Dažniausia herpes zoster virusinės infekcijos neurologinė komplikacija yra poherpetinė neuralgija. Herpes zoster gali būti poherpetinio galvos skausmo, galūnės parezės priežastis. Epilepsiniai priepuoliai ir encefalopatija yra dažniausios influenza virusinės infekcijos neurologinės komplikacijos. Pateikti klinikiniai atvejai, atskleidžiantys herpes ir influenza virusinių infekcijų sukeltų neurologinių komplikacijų įvairovę. Apžvelgta naujausia mokslinė literatūra apie neurologinę patologiją, sergant herpes ir influenza virusinėmis infekcijomis.

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