Die Gehirngefäße beim Hochdruck

Anders, H. E.; Eicke, Werner J.

doi:10.1007/bf01813840

Cited by 29 publications

(2 citation statements)

References 16 publications

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“…4a) and small branches of the spinal grey matter usually associated with severe generalized atherosclerosis and systemic hypertension. Contrary to the findings of Arendt and Wunscher (1954) we never were able to detect senile 'kongophilic' or 'drusige' angiopathy (Schlote, 1965) and genuine hyalinosis, i.e., hypertensive angiopathy (Anders and Eicke, 1940) in the spinal cords examined.…”

Section: Resultscontrasting

confidence: 99%

Spinal cord arteriosclerosis and progressive vascular myelopathy.

Jellinger¹

1967

Journal of Neurology, Neurosurgery & Psychiatry

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Section: Resultscontrasting

confidence: 99%

Spinal cord arteriosclerosis and progressive vascular myelopathy.

Jellinger¹

1967

Journal of Neurology, Neurosurgery & Psychiatry

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“…Light microscopy of human cases has shown that colla gens are major constituents of hyaline [8, [52][53][54] which is in line with our immunohistochemical observations indi cating the presence of collagens type 1, II and V as well as basal lamina components in the hyaline material. It is not known to what extent other ECM components and plas ma proteins occur in arterioles presenting hyalinosis.…”

Section: A Neriolar 11\>a Linos Issupporting

confidence: 79%

Structural and Vasoactive Factors Influencing Intracerebral Arterioles in Cases of Vascular Dementia and Other Cerebrovascular Disease: A Review

Zhang

Badonic²,

Höög³

et al. 1994

Dement Geriatr Cogn Disord

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The condition of the brain parenchyma in cases of vascular dementia and other cerebrovascular conditions may be influenced by structural and functional changes of the terminal intracerebral blood vessels. Arterioles can develop obliterative lesions, capillaries and postcapillary venules can be altered, causing edema. The first part of this review is focused on expression of different types of collagens and other components of the extracellular matrix in intracerebral arterioles. The changes present in hereditary multi-infarct disease of the brain are compared with those occurring in the Binswanger type of encephalopathy and cases presenting hyalinosis of intracerebral vessels. Deposition of collagens in degenerated parts of the media and adventitia of the arterioles may contribute to impaired blood flow regulation in the brain parenchyma. Fibrillary collagens and basal laminae are probably the most important components of the hyaline material in vessels showing ‘hyalinosis’. The second part of our review concerns the possibility that the vasoactive peptide, endothelin-1, released from reactive astrocytes, can influence the function of intracerebral arterioles. Normal astrocytes do not show endothelin-1-like immunoreactivity, but in cases of infarcts, lacunes, hereditary multi-infarct disease, Binswanger''s encephalopathy and Alzheimer''s disease numerous reactive astrocytes express such immunoreactivity. If endothelin-1 is produced and released from reactive astrocytes it may reach intracerebral arterioles and induce long-lasting vasoconstriction. Endothelin-1 is the most powerful vasoconstrictor peptide known to date and has mitogenic capacity. It may promote cellular mechanisms leading to astrocytic gliosis and neovascularization.

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Die koronarstenotisch verursachte akute Koronarinsuffizienz

Buchner

1975

Hypoxie

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Die Gehirngefäße beim Hochdruck

Cited by 29 publications

References 16 publications

Spinal cord arteriosclerosis and progressive vascular myelopathy.

Spinal cord arteriosclerosis and progressive vascular myelopathy.

Structural and Vasoactive Factors Influencing Intracerebral Arterioles in Cases of Vascular Dementia and Other Cerebrovascular Disease: A Review

Die koronarstenotisch verursachte akute Koronarinsuffizienz

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