Spinal cord arteriosclerosis and progressive vascular myelopathy.

Syringomyelia as a sequel to traumatic paraplegia

“…Average length of follow-up since onset of syringomyelia was 6 years ( Table I). None of these patients had neuro pathic joints, bilaterality or brain stem involvement which have been reported (Jellinger, 1967;Rossier, Werner, Wildi & Berney, 1968;Barnett et ai., 1976).…”

“…Lesions remote from the primary cord injury have been reported apparently related to arterial injury. The role of ischaemia in producing small areas of infarction followed by cavitation has been suggested (Mair & Druckman, 1953;Jellinger, 1967). Recently there have been reports dealing with cyst formation on a biochemical basis and there is evidence that lysosomes and other cellular enzymes play a role in cyst formation after trauma Kao, Chang & Bloodworth, 1977).…”

Servikal miyelopati ve radikulopati; ayırıcı tanı

“…Farinks ve üst mediastenin diğer lezyonları, omurgaya, brakiyal pleksusa ve göğüs kafesine invaze olabilir. [18] Kafa içi lezyonlar, servikal radiküler ağrıları taklit edebilir. Hastada üst motor nöron semptomları varsa, muayene eden kişi beyni sorunların kaynağı olarak düşünmelidir.…”

Atraumatic ischaemic myelopathy

“…INFARCTION of the spinal cord has been considered to be rare when compared with cerebral infarction (Garland et at., 1966) which may be due to the fact that the frequency of arteriosclerotic changes in major spinal arteries occur in 2·2-10 per cent of cases compared with cerebrovascular arteriosclerotic changes in 39 per cent of the population (Jellinger, 1967). Although Bastian in 1910 was first to suggest that spinal cord softening might be a consequence of vascular occlusion but for several years the number of well documented cases of ischaemic myelopathy remained small and often anecdotal with syphilitic vasculitis.…”