Continuity of leanness/obesity from childhood to adolescence.

Nakatsuka, Haruo; Kasahara, Mikio; Watanabe, Takao; Ikeda, Masayuki

doi:10.1620/tjem.157.381

Cited by 3 publications

(4 citation statements)

References 30 publications

(34 reference statements)

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“…When assessed cross‐sectionally, a very thin person could perhaps just be ill, be on a weight‐reducing diet, or be likely to gain a great deal of weight in the future. Nakatsuka et al (42) tracked both extreme thinness and extreme obesity in children across the ages of 11, 14 and 18, and found that both conditions were stable, although some regression to the mean was observed. Of particular note is that no children who were extremely thin became obese, or vice versa, during the period of observation.…”

Section: Evidence Supporting Thinness As a Unique Phenotypementioning

confidence: 99%

The genetic epidemiology of thinness

Bulik

Allison

2001

Obesity Reviews

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Most genetic research in the area of human obesity asks the question 'Why are certain people obese?' Considerably less attention has been paid to the question of why certain people are not obese, particularly given the obesogenic environment that permeates the western culture. We present data from human and animal studies and evolutionary arguments supporting the notion that genetic studies of thinness or obesity resistance may yield important and complementary findings to genetic studies of obesity. We offer strategies for further refining the definition of thinness, weigh the advantages and disadvantages of potential sampling strategies and suggest candidate genes for thinness or obesity resistance.

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Section: Evidence Supporting Thinness As a Unique Phenotypementioning

confidence: 99%

The genetic epidemiology of thinness

Bulik

Allison

2001

Obesity Reviews

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“…The national school health data on the body habitus of children are based on the stratified sampling at a rate of 3 to 4% (Ministry of Education, Science and Culture 1989). Such analysis of a topographic scale as the present study is hardly possible unless each and all children are examined like the October measurement done in the city of Sendai (Kondo et al 1978;Ikeda et al 1983;Nakatsuka et al 1989). …”

Section: Discussionmentioning

confidence: 96%

“…The body habitus of children in the present day society is an integrated expression of both genetic and environmental factors (Annest et al 1983 Nakatsuka et al 1989), and much more complex than it was in 1950's when a single factor of nutrition played almost a decisive role (Kondo et al 1978). In fact, nutritional surveys conducted in the vicinity of the present study regions proved that women in agricultural areas take nutrients as sufficient as their peers in urban areas (Ikeda et al 1988;Nakatsuka et al 1988b).…”

Section: Discussionmentioning

confidence: 99%

Possible urban-rural difference in habitus of school children in Sendai.

Nakatsuka

Satoh

Watanabe

et al. 1990

Tohoku J. Exp. Med.

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“…Height and weight were measured at the pre-and posttreatment timepoints to calculate the Rohrer index. This index, which is commonly used to evaluate the physical constitution of Japanese schoolchildren, 32 was calculated as body weight (g)/height (cm 3 ). The times of sleep onset and offset, total sleep duration, number and duration of the periods in which patients awoke from sleep, and number and duration of napping periods were assessed using sleep logs spanning a period from 1 week before and for 1 week after the treatment.…”

Section: Examinationmentioning

confidence: 99%

Serum concentrations of insulin-like growth factor-1 as a biomarker of improved circadian rhythm sleep-wake disorder in school-aged children

Kimura

Toyoura²,

Toyota³

et al. 2020

Journal of Clinical Sleep Medicine

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Study Objectives: We aimed to investigate whether improvements in the symptoms of circadian rhythm sleep-wake disorder after treatment were associated with an increase in serum insulin-like growth factor-1 (IGF-1) concentration. Methods: Eighty-seven school-aged children (32 males, 55 females), aged 14.31 ± 1.50 years (mean ± standard deviation), who were admitted to our hospital with circadian rhythm sleep-wake disorder received treatment for 6-8 weeks consisting of the following protocol: (1) lights-out for sleep occurred at 21:00, (2) phototherapy for waking started at 06:00 or 07:00, and (3) light exercise was required every day (eg, a 20-to 30-minute walk). Blood samples were collected at 08:00 AM to measure the serum concentrations of IGF-1, pre-and posttreatment. Results: The mean times of day of sleep onset and offset at the pre-and posttreatment timepoints were 23:32 ± 4.21 and 10:27 ± 2.98, and 21:26 ± 0.55 and 06:50 ± 0.70, respectively. The mean times of day of sleep onset and offset measured at the posttreatment timepoint were significantly earlier compared with the pretreatment baselines (P <.01). The mean serum levels of IGF-1 significantly increased from 315.59 ± 68.26 ng/mL at pretreatment to 335.09 ± 69.78 ng/mL at posttreatment (P < .01). Conclusions: Improvements in the symptoms of patients with circadian rhythm sleep-wake disorders were associated with increased serum concentrations of IGF-1, suggesting that serum IGF-1 may be a biomarker of improvements in school-aged children with circadian rhythm sleep-wake disorder.

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Continuity of leanness/obesity from childhood to adolescence.

Cited by 3 publications

References 30 publications

The genetic epidemiology of thinness

The genetic epidemiology of thinness

Possible urban-rural difference in habitus of school children in Sendai.

Serum concentrations of insulin-like growth factor-1 as a biomarker of improved circadian rhythm sleep-wake disorder in school-aged children

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