Coffee consumption and risk of coronary heart diseases: A meta-analysis of 21 prospective cohort studies

Wu, Jiang‐Nan; Ho, Suzanne C.; Zhou, Chun; Liu, Wenhua; Chen, Weiqing; Wang, Cuiling; Chen, Yuming

doi:10.1016/j.ijcard.2008.06.051

Cited by 162 publications

(134 citation statements)

References 62 publications

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“…The effect does not appear to be specific to light, moderate or heavy coffee drinking. This result is consistent with current meta analyses on the coffee consumption and risk of CHD, which indicate that habitual moderate coffee drinking is associated with a lower risk of CHD in women [7], but has no effect on men [6]. These findings suggest that coffee drinking may be one of the healthy lifestyles, especially for women, which could reduce the risk of stroke and CHD.…”

Section: Discussionsupporting

confidence: 91%

“…It is difficult to make precise definitions of light, moderate and heavy coffee consumption. We adopted the principles that used in a meta analysis on relationship between coffee consumption and coronary heart disease [7]. Coffee drinking was divided into three groups: light, moderate, and heavy for the subgroup analysis.…”

Section: Discussionmentioning

confidence: 99%

“…It indicates that coffee consumption could decrease the risk of Type 2 diabetes [5]. Meta analysis on coffee consumption and coronary heart disease (CHD) shows that coffee can reduce CHD risk in women, but may have no effect in men [6,7]. There are only a few researches about the correlation between coffee consumption and stroke.…”

Section: Introductionmentioning

confidence: 99%

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Coffee consumption and risk of stroke: a meta-analysis of cohort studies

Rui¹,

Wang

Song

et al. 2012

Open Medicine

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Section: Discussionsupporting

confidence: 91%

Section: Discussionmentioning

confidence: 99%

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Coffee consumption and risk of stroke: a meta-analysis of cohort studies

Rui¹,

Wang

Song

et al. 2012

Open Medicine

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“…A sizable body of evidence is available on intakes of specific beverages such as alcohol, coffee, tea, and sugar-sweetened beverages in relation to a variety of health outcomes (4)(5)(6)(7)(8)(9)(10). The association of water intake and body-weight management has also received considerable attention (11).…”

Section: Introductionmentioning

confidence: 99%

A prospective study of water intake and subsequent risk of all-cause mortality in a national cohort

Kant

Graubard

2017

The American Journal of Clinical Nutrition

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Background: Water, an essential nutrient, is believed to be related to a variety of health outcomes. Published studies have examined the association of fluid or beverage intake with risk of mortality from coronary diseases, diabetes, or cancer, but few studies have examined the association of total water intake with all-cause mortality. Objective: We examined prospective risk of mortality from all causes in relation to intakes of total water and each of the 3 water sources. Design: We used public-domain, mortality-linked water intake data from the NHANES conducted in 1988-1994 and 1999-2004 for this prospective cohort study (n = 12,660 women and 12,050 men; aged $25 y). Mortality follow-up was completed through 31 December 2011. We used sex-specific Cox proportional hazards regression methods that were appropriate for complex surveys to examine the independent associations of plain water, beverage water, water in foods, and total water with multiple covariate-adjusted risk of mortality from all causes. Results: Over a median of 11.4 y of follow-up, 3504 men and 3032 women died of any cause in this cohort. In men, neither total water intake nor each of the individual water source variables (plain water, water in beverages, and water in foods) was independently related with risk of all-cause mortality. In women, risk of mortality increased slightly in the highest quartile of total or plain water intake but did not approach the Bonferroni-corrected level of significance of P , 0.002. Conclusions: There was no survival advantage in association with higher total or plain water intake in men or women in this national cohort. The slight increase in risk of mortality noted in women with higher total and plain water intakes may be spurious and requires further investigation.Am J Clin Nutr 2017;105:212-20.

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“…An adverse effect between coffee drinking and cardiovascular disease risk was found by Cornelis and El-Sohemy (2007). In contrast, Wu et al (2009) in a meta-analysis of a number of cohort studies, found coffee drinking decreased the risk of cardiovascular disease. Chlorogenic acids are important components of coffee and are considered to be responsible for the beneficial effects of coffee, while caffeine is believed to mediate a number of harmful effects.…”

Section: Introductionmentioning

confidence: 95%

Green Coffee Extract Protects H9C2 Cardiomyocytes from Doxorubicin Induced Apoptosis

Malkapuram¹,

Venkataram²,

Rucha³

et al. 2016

Research J. of Medicinal Plant

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Green Coffee Extract (GCE), an extract of Coffea arabica bean is a popular health supplement employed for anti-obesity and anti-diabetic effects. Here a hydroalcoholic extract of Green Coffee (GCE) was evaluated for its potential as a cardioprotective agent against Doxorubicin (Dox) induced cardiac insult in a H9C2 rat cardiomyocyte in vitro model system. The GCE was tested in an MTT viability assay using 1 μM Dox with and without GCE pretreatment at 50, 100 and 250 μg mLG 1 concentrations. GCE was also tested for its free radical scavenging ability in a DPPH assay at 10 concentrations (500 μg mLG 1 maximum concentration). To understand the mechanism of action of cardioprotection, mitochondrial membrane potential (Δψm) was compared between Dox treated cells with and without GCE pretreatment, using the JC-1 dye. Finally, the activation of caspase-3/7 was quantitated. Findings from the above experiments demonstrated that GCE rescued H9C2 cardiomyocytes from Dox induced loss of cell viability in a dose-dependent manner. While Dox treatment caused a clear decrease in the JC-1 ratio from 2 to 1.6 due to loss of Δψm, pretreatment with GCE at 25, 50 and 100 μg mLG 1 restored the JC-1 ratio to 1.6, 1.9 and 2.0, respectively. Dox treatment potently induced caspase 3/7 activity by 5 fold and pre-treatment with GCE at 100 and 500 μg mLG 1 reduced this activation to 3.5 and 1.5 fold, respectively. This data clearly demonstrates that GCE is strongly cardioprotective against Dox induced cardiac insult and the mechanism of action is by blocking activation of intrinsic apoptotic pathway.

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Coffee consumption and risk of coronary heart diseases: A meta-analysis of 21 prospective cohort studies

Cited by 162 publications

References 62 publications

Coffee consumption and risk of stroke: a meta-analysis of cohort studies

Coffee consumption and risk of stroke: a meta-analysis of cohort studies

A prospective study of water intake and subsequent risk of all-cause mortality in a national cohort

Green Coffee Extract Protects H9C2 Cardiomyocytes from Doxorubicin Induced Apoptosis

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