“…In Sendai virus-specific CTL, coincidence in the H-2K region seems important rather than that in the H-2D region (13,25,34,35,39,41,59). Several reports demonstrated that inbred mice of the H-2k haplotype were low responders to secondary in vitro stimulation of CTL (14,40,53), whereas other investigations showed that H-2k mice generated normal CTL responses (5,7,16,24,37,39,61). The present data indicated that CTL could be elicited in H-2k-bearing C3H/He mice by intranasal infection with TR-2 and a secondary CTL response could be stimulated in vivo by reinfection with wild-type Sendai virus.…”