Cardiac pathology in death from electrocution

Fineschi, Vittorio; Karch, Steven B.; D’Errico, Stefano; Pomara, Cristoforo; Riezzo, Irene; Turillazzi, Emanuela

doi:10.1007/s00414-005-0011-8

Cited by 51 publications

(27 citation statements)

References 32 publications

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“…In most clinical cases, heart failure is a consequence of cardiovascular disease. However, in medicolegal situations, heart failure may be attributable to other causes, and its identification could have important legal implications [2][3][4][5][6][7][8]. It could be associated with acute disease or trauma, with drug intoxication or a protracted state of agony.…”

Section: Introductionmentioning

confidence: 97%

Evaluation of postmortem measurement of NT-proBNP as a marker for cardiac function

et al. 2008

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Clinical biomarkers of cardiac function could also be monitored postmortem. Among the natriuretic peptides, the aminoterminal portion of pro-brain natriuretic peptide (NT-proBNP) appears to be a more reliable postmortem tool than the BNP, owing to its longer half-life and greater stability. In living persons, NT-proBNP is considered to be a marker of heart failure, and its level rises after cardiac ischemia. The goal of this study was first to evaluate the postmortem stability of NT-proBNP, then to measure the NT-proBNP levels in postmortem cases of heart failure related to coronary ischemia. The goal of this study was also to evaluate the correlations between different specimens collected at autopsy (e.g. blood, serum, vitreous humor and pericardial fluid). The study included 96 cases, which were classified into 4 groups according to the autopsy and histological findings. The NT-proBNP levels were significantly higher in individuals who had suffered from chronic cardiac ischemia, with or without acute coronary events, than in either control cases or those who had suffered from acute thromboembolism or acute rupture of a plaque without chronic cardiac ischemia. The highest levels were registered in individuals who had suffered from acute coronary thromboembolism in association with chronic coronary ischemia. Good correlations in the NT-proBNP levels for the different specimens were observed between samples of femoral blood, serum, and pericardial fluid. Our data indicated that postmortem measurements of NT-proBNP are reliable and compatible with clinical findings.

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Section: Introductionmentioning

confidence: 97%

Evaluation of postmortem measurement of NT-proBNP as a marker for cardiac function

et al. 2008

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“…Sicher ist jedoch, dass sich die todesursächliche Wirkung des Stroms am Herzen entfaltet; Kammerflimmern oder Asystolie bewirken den Herztod [2,9,14,20,23,25,26]. Hyperkontraktionsbanden und -nekrosen sowie myozelluläre Segmentierungen infolge der heftigen, teils tetanischen Kontraktion der Myozyten sind dokumentiert [12,14].…”

Section: Elektrotechnische Untersuchungen Des Leichenfundortesunclassified

Interdisziplinäre Aufklärung eines ungewöhnlichen Stromunfalls

Heitkötter

Schulz

Koch³

et al. 2015

Rechtsmedizin

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Makromorphologische und histologische Befunde zum Nachweis eines Stromtodes sind oft rar und nicht spezifisch. Daher kann es im Einzelfall schwierig sein, sich ohne weitere Anhaltspunkte bei entsprechenden Verdachtsfällen eindeutig auf die Einwirkung elektrischen Stroms als Todesursache festzulegen. Neben der klassischen morphologischen Analyse bedarf es daher häufig der Mithilfe physikalischer Verfahren sowie der Begutachtung des Leichenfundorts durch einen technischen Sachverständigen.

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“…Mekanisme ini dimungkinkan oleh karena perubahan potensial listrik pada pacu jantung dan secara berurutan akan tersebar melalui sistem hantaran ke seluruh bagian jantung. 7 Jantung terdiri atas jaringan yang mempunyai sifat kelistrikan yang tinggi. Berbeda dengan sel saraf dan otot rangka yang membrannya tetap berada pada potensial istirahat yang konstan kecuali apabila dirangsang, sel otot ritmik jantung tidak memiliki potensial istirahat.…”

Section: Pendahuluanunclassified

“…Kurang lebih 99% otot jantung (miokardium) adalah sel kontraktif yang berkontraksi. 7 Terdapat peningkatan ketebalan miokardium rata-rata dimulai dari kelompok kontrol sampai dengan kelompok perlakuan 3. Setelah dilakukan uji one way ANOVA didapatkan p=0,019.…”

Section: Pembahasanunclassified

Peningkatan Ketebalan Miokardium Mencit (Mus musculus L.) Akibat Paparan Medan Listrik Tegangan Tinggi

Busman¹,

Muhartono²

2013

mkb

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AbstrakPembangunan saluran transmisi listrik tegangan tinggi diduga dapat merugikan manusia atau makhluk hidup lain. Tujuan penelitian untuk mengetahui pengaruh paparan medan listrik tegangan tinggi pada ketebalan miokardium ventrikel kiri mencit jantan (Mus musculus L.). Penelitian dilaksanakan di Laboratorium Zoologi Fakultas Matematika dan Ilmu Pengetahuan Alam Universitas Lampung serta Laboratorium Patologi Balai Penyidikan dan Pengujian Veteriner Regional III Bandar Lampung, pada bulan Juni−November 2011.Penelitian menggunakan rancangan acak lengkap dengan 4 perlakuan, ulangan 6 kali, dan dibagi dalam empat kelompok. Kelompok kontrol (K) tidak diberi perlakuan, kelompok 1 (P1) diberi paparan 5 kV/m, kelompok 2 (P2) diberi paparan 6 kV/m, dan kelompok 3 (P3) diberi paparan 7 kV/m, masing-masing 8 jam/hari selama 37 hari. Data dianalisis menggunakan analysis of variance. Hasil penelitian didapatkan ketebalan miokardium ventrikel kiri mencit jantan pada K sebesar 1.329,83±173,29 µm; P1 sebesar 1.507,50±109,24 µm; P2 sebesar 1.536,70±103.42 µm; dan P3 sebesar 1.574.23±123,36 µm. Terdapat peningkatan ketebalan miokardium rata-rata dengan bertambahnya daya paparan medan listrik (p=0,019). Simpulan, terdapat hubungan antara paparan medan listrik tegangan tinggi dan perubahan ukuran ketebalan miokardium ventrikel kiri mencit jantan, semakin tinggi paparan medan listrik semakin tebal miokardium ventrikel kiri mencit jantan. Research using completely randomized design with 4 treatments, replicated 6 times and divided into four groups.The control group (K) was not given treatment, group 1 (P1) given exposure to 5 kV/m, group 2 (P2) given exposure to 6 kV/ mand group 3 (P3) given exposure to 7 kV/m for 8 hours/day, to 37 days. Data were analyzed using analysis of variance. The results obtained thickness of the left ventricular myocardium of male mice at K1,329.83±173.29 μm; P1 at 1,507.50±109.24 μm; P2 at 1,536.70±103.42 μm, and P3 at 1,574.23±123.36 μm. There was an increase in the average thickness of the myocardium with increasing exposure to power an electric field with a statistical test obtained (p=0.019). In conclusion, there is a significant relationship between exposure to high-voltage electric field to change the size of the thickness of the left ventricular myocardium male mice, the higher the electric field exposure thicker left ventricular myocardium male mice. [MKB. 2013;45(3):155-60]

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Cardiac pathology in death from electrocution

Cited by 51 publications

References 32 publications

Evaluation of postmortem measurement of NT-proBNP as a marker for cardiac function

Evaluation of postmortem measurement of NT-proBNP as a marker for cardiac function

Interdisziplinäre Aufklärung eines ungewöhnlichen Stromunfalls

Peningkatan Ketebalan Miokardium Mencit (Mus musculus L.) Akibat Paparan Medan Listrik Tegangan Tinggi

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