Bilateral temporalis hypertrophy

Ali, Kausar; Sittampalam, G.; Malik, M.A.

doi:10.1016/j.ijom.2009.09.005

Cited by 4 publications

(7 citation statements)

References 9 publications

(7 reference statements)

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“…Usualmente a forma adquirida é resultado de hábitos mandibulares parafuncionais tais como bruxismo e hiperfunção mastigatória. 1 O diagnóstico diferencial inclui desde condições inflamatórias até neoplasias. São elas: miopatia inflamatória, miosite proliferativa, distrofia muscular, lipomatose, hemangioma cavernoso, lipossarcoma, rabdomiossarcoma, leucemia infiltrativa e linfoma.…”

Section: Discussionunclassified

“…Uma condição pouco frequente, mas que na maioria dos casos pode comprometer negativamente o contorno craniofacial, consiste na Hipertrofia Idiopática do Músculo Temporal, eventualmente associada à hipertrofia da musculatura massetérica uni ou bilateralmente. 1 Apresentamos o caso de uma paciente jovem, com queixas estéticas de proeminência bitemporal há aproximadamente 10 anos.…”

Section: Introductionunclassified

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Hipertrofia idiopática bilateral de músculos temporal e masseter / Bilateral idiopathic hypertrophy of temporal and masseter muscles

Tonello

Bastos²,

Feitosa

et al. 2021

BASR

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A hipertrofia bilateral do músculo temporal é incomum e pode estar associada ao comprometimento de outros músculos do sistema mastigatório, como masseter e pterigóideo. Além de possíveis repercussões na estética facial, há relatos de disfunção da articulação temporomandibular, como dificuldade de mastigação, dor e trismo. O tratamento pode ser feito de forma conservadora, através de medidas não-invasivas ou por meio de procedimentos cirúrgicos. O presente trabalho objetiva trazer uma revisão da literatura e relatar o caso de uma paciente jovem com queixas exclusivamente estéticas relacionadas à proeminência bitemporal, diagnosticada com hipertrofia temporal e massetérica, e sua condução terapêutica.Palavras-chave: Hipertrofia, transtornos da articulação temporomandibular, anormalidades musculoesqueléticas músculo temporal, músculo Masseter.

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Section: Discussionunclassified

Section: Introductionunclassified

Hipertrofia idiopática bilateral de músculos temporal e masseter / Bilateral idiopathic hypertrophy of temporal and masseter muscles

Tonello

Bastos²,

Feitosa

et al. 2021

BASR

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“…In the case reported, we could not identify any parafunctional habit or psychosocial disturbance and for that reason, the muscle hypertrophy may be regarded as idiopathic. Traditionally, surgical procedures have beenthe treatment of choice for reduction of masticatory muscle hypertrophy [4,9,10]. The first surgical technique used to treat the masseteric hypertrophy was published in 1947 and consists in partial resection of the muscle bulk through extra-oral approach [9].…”

Section: Discussionmentioning

confidence: 99%

“…The botulinum toxin A has been used successfully to correct temporalis and masseteric hypertrophy [10]. It is an established drug for the treatment of a variety of neuromuscular disorders and its effect of paralysis and subsequent muscle atrophy has been beneficial to treat this condition [17].…”

Section: Open Accessmentioning

confidence: 99%

“…It is an established drug for the treatment of a variety of neuromuscular disorders and its effect of paralysis and subsequent muscle atrophy has been beneficial to treat this condition [17]. However, these effects are temporary and, for that reason, hypertrophic muscles could regain their original size after few months [10,17,18]. It can be a non-invasive option to treatpatients with muscle hypertrophy, butthis therapy has showed a variable rate of recurrence and lack of long-term follow-up studies.…”

Section: Open Accessmentioning

confidence: 99%

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Surgical treatment of bilateral temporalis and masseteric hypertrophy: Report of a case

Parente¹,

Silvares²,

Naegele³

et al. 2013

OJST

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Hypertrophy of the masticatory muscles is characterized by generalized enlargement of the muscular tissue that affects the facial esthetic and may or not be accompanied by pain [1-3]. This condition can be congenital, but more often it is acquired. The origin has been attributed to muscle hyperactivity and parafunctionsoriginated from stressful lifestyle that causes bruxing or clenching [2,3]. Masseteric muscle hypertrophy is a relative common clinical entity that can affect one or both sides and is also thought to causesecondary enlargement of the mandibular angle as a result of functional remodeling. Temporalis muscle hypertrophy is a rare clinical entity and only a few cases are reported. More often, it presents a bilateral involvement and is usually associated with masseteric hypertrophy [3]. The aim of this report is to present a case of bilateral temporalis and masseteric muscles hypertrophy treated surgically.

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Antioxidant effect of human placenta hydrolysate against oxidative stress on muscle atrophy

Bak

et al. 2018

Journal Cellular Physiology

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Sarcopenia, which refers to the muscle loss that accompanies aging, is a complex neuromuscular disorder with a clinically high prevalence and mortality. Despite many efforts to protect against muscle weakness and muscle atrophy, the incidence of sarcopenia and its related permanent disabilities continue to increase. In this study, we found that treatment with human placental hydrolysate (hPH) significantly increased the viability (approximately 15%) of H O -stimulated C2C12 cells. Additionally, while H O -stimulated cells showed irregular morphology, hPH treatment restored their morphology to that of cells cultured under normal conditions. We further showed that hPH treatment effectively inhibited H O -induced cell death. Reactive oxygen species (ROS) generation and Mstn expression induced by oxidative stress are closely associated with muscular dysfunction followed by atrophy. Exposure of C2C12 cells to H O induced abundant production of intracellular ROS, mitochondrial superoxide, and mitochondrial dysfunction as well as myostatin expression via nuclear factor-κB (NF-κB) signaling; these effects were attenuated by hPH. Additionally, hPH decreased mitochondria fission-related gene expression (Drp1 and BNIP3) and increased mitochondria biogenesis via the Sirt1/AMPK/PGC-1α pathway and autophagy regulation. In vivo studies revealed that hPH-mediated prevention of atrophy was achieved predominantly through regulation of myostatin and PGC-1α expression and autophagy. Taken together, our findings indicate that hPH is potentially protective against muscle atrophy and oxidative cell death.

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Bilateral temporalis hypertrophy

Cited by 4 publications

References 9 publications

Hipertrofia idiopática bilateral de músculos temporal e masseter / Bilateral idiopathic hypertrophy of temporal and masseter muscles

Hipertrofia idiopática bilateral de músculos temporal e masseter / Bilateral idiopathic hypertrophy of temporal and masseter muscles

Surgical treatment of bilateral temporalis and masseteric hypertrophy: Report of a case

Antioxidant effect of human placenta hydrolysate against oxidative stress on muscle atrophy

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