A Case Report on the Change of the Refractive Power After a Blunt Trauma

Abstract

PurposeTo determine the pathogenesis of transient myopia after blunt eye trauma.MethodsIn one patient, the refraction of both eyes (the left eye was injured, but the right eye was not) was measured with an autorefractometer. The cycloplegic refraction was measured at the early stage of trauma and again 3 months after the blunt eye injury. The angle and depth of the anterior chamber, the ciliary body, and the choroids were examined by ultrasound biomicroscopy (UBM) over 3 months. The depth of the anterior chamber, the thickness of the lens, and the axial length were measured by A-scan ultrasonography in both eyes. During the 3 months after the injury, we made comparisons between the menifest and the cycloplegic refractions, the depths of anterior chambers, the thickness of the lenses, the axial lengths, and the UBM-determined appearances of the angles and depths of the anterior chambers, the ciliary bodies, and the choroids in both eyes.ResultsWe suspect that the depth reduction in the anterior chamber, the increase in anterior to posterior lens diameter, and the edema in the ciliary body are all related to the change in the refractive power following the blunt trauma.ConclusionsUltrasound biomicroscopy (UBM) and ultrasonography of the anterior segment in the eye may be helpful to diagnose and confirm changes in the refractive power that occur after trauma.

Evaluation of Refractive Power Changes after Soccer Ocular Trauma in a Child Eight Years Old

“…7,[9][10] In our case we also diagnosed ciliar body edema with AS-OCT. Despite many previous studies describing the myopic changes of blunt ocular trauma, its effect on astigmatism is unclear.…”

“…[1][2][3][4] Transient myopia is the most common refractive problem after blunt ocular trauma, luckily it resolves within weeks. [5][6][7] Myopia may be caused by ciliary body edema and/or thickening of the crystalline lens. Blunt ocular trauma can also induce astigmatism.…”

Myopie transitoire post-traumatique ? À propos d’un cas

“…Un bilan clinique et paraclinique, notamment échographique, nous a permis de proposer les principaux mécanismes responsables de cette myopisation : bascule antérieure du plan iridocristallinien, augmentation du diamètre antéropostérieur du cristallin, décollement ciliochoroïdien, oedème du corps ciliaire [1]. La bascule antérieure du plan iridocristallinien a été objectivé par tous les examens, notamment la biométrie du segment antérieur au Lenstar LS900, l'OCT-SA et l'UBM.…”

Induced Myopia Secondary to Blunt Trauma

“…It is sudden onset and usually transient, recovering within a few weeks after the trauma, although some cases may be long-standing [2]. Possible etiologic causes for this condition are as follows: spasm of the ciliary body, increased crystalline lens effective power secondary to its forward shift, ciliochoroidal effusion causing forward displacement of the crystalline lens-iris diaphragm, axial thickening of the natural lens, and other sources of choroidals which could undoubtedly produce the same refractive issues such as varicella infection [2][3][4]. Although the closed head trauma may produce decreased visual acuity secondary to the bilateral spasm of accommodation, one case was reported with persistent myopia which was still present three months after trauma.…”