In this paper, the stochastic stability of networked control systems (NCSs) with nonlinear perturbation and parameter uncertainties is studied. A decentralized dynamic event triggering scheme (DDETS) is introduced to reduce energy consumption and network transmission burden. Each channel can decide whether to transmit signals sampled by corresponding sensors through this mechanism. Due to the unreliability of communication network, a new mathematical model of NCSs based on multi‐channel fading and hybrid cyber attacks is established, and the actuator is constrained by saturation. A sufficient condition for stochastic stability based on static controller is derived through Lyapunov stability theory, and two numerical examples are given to demonstrate the effectiveness of the method.
Experimental studies have shown that astrocytes participate in epilepsy through inducing the release of glutamate. Meanwhile, considering the disinhibition circuit among inhibitory neuronal populations with different time scales and the feedforward inhibition connection from thalamic relay nucleus to cortical inhibitory neuronal population, here, we propose a modified thalamocortical field model to systematically investigate the mechanism of epilepsy. Firstly, our results show that rich firing activities can be induced by astrocyte dysfunction, including high or low saturated state, high- or low-frequency clonic, spike-wave discharge (SWD), and tonic. More importantly, with the enhancement of feedforward inhibition connection, SWD and tonic oscillations will disappear. In other words, all these pathological waveforms can be suppressed or eliminated. Then, we explore the control effects after different external stimulations applying to thalamic neuronal population. We find that single-pulse stimulation can not only suppress but also induce pathological firing patterns, such as SWD, tonic, and clonic oscillations. And we further verify that deep brain stimulation can control absence epilepsy by regulating the amplitude and pulse width of stimulation. In addition, based on our modified model, 3 : 2 coordinated reset stimulation strategies with different intensities are compared and a more effective and safer stimulation mode is proposed. Our conclusions are expected to give more theoretical insights into the treatment of epilepsy.
This paper investigates the problem of adaptive event-triggered synchronization for uncertain FNNs subject to double deception attacks and time-varying delay. During network transmission, a practical deception attack phenomenon in FNNs should be considered; that is, we investigated the situation in which the attack occurs via both communication channels, from S-C and from C-A simultaneously, rather than considering only one, as in many papers; and the double attacks are described by high-level Markov processes rather than simple random variables. To further reduce network load, an advanced AETS with an adaptive threshold coefficient was first used in FNNs to deal with deception attacks. Moreover, given the engineering background, uncertain parameters and time-varying delay were also considered, and a feedback control scheme was adopted. Based on the above, a unique closed-loop synchronization error system was constructed. Sufficient conditions that guarantee the stability of the closed-loop system are ensured by the Lyapunov-Krasovskii functional method. Finally, a numerical example is presented to verify the effectiveness of the proposed method.
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