The brain has the ability to protect itself against ischemia when stimulated. The elucidation of this mechanism enables the application of preconditioning inducing substances (some anesthetics), other drugs, and non-pharmacological measures, such as hypothermia, aimed at inducing tolerance to ischemic lesions.
RESUMONunes RR, Cavalcante SL -Influência da Dexmedetomidina na Concentração Expirada do Sevoflurano. Avaliação pelo Índice Bispectral, Taxa de Supressão e Análise Espectral da Potência do Eletroencefalograma GII foi 6,35 ± 0,93 minutos (p < 0,05). O tempo de alta hospitalar no GI foi 7,45 ± 0,69 horas e no GII foi 8,37 ± 0,88 horas (p < 0,05). Groups I and II was 11 ± 0.91 min. and 6.35 ± 0.93 min., respectively (p < 0.05). Time for hospital discharge was 7. 45 ± 0.69 h in Group I and 8.37 ± 0.88 h in Group II (p < 0.05
Justificativa e Objetivos -A dexmedetomidina, um a 2 -agonista adrenérgico, tem sido descrita como capaz de reduzir o consumo tanto de agentes venosos como inalatórios. O o b j e t i v o d e s t e e s t u d o f o i a v a l i a r a i n f l u ê n c i a d a
Conclusões
Anesthetic techniques investigated in this study showed a similar pattern, with no increase in metabolic substrates and oxidative stress during surgical correction of congenital heart defects in non-cyanotic children.
Sedation levels showed significant correlation with the increase in ketamine S(+) dosage. However, increased BIS levels may have reflected increased pEMG induced by ketamine S(+).
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