. How to cite this article: Kharde KR, Soujanya S (2014) Effect of garlic and neem leaf powder supplementation on growth performance and carcass traits in broilers, Veterinary World 7(10): 799-802. AbstractAim: Present experiment was designed to study the effect of supplementation of garlic and neem leaf powder (NLP) on growth performance and carcass yields in broilers. Materials and Methods:A total of 210-day-old commercial male broiler chicks (Ven Cobb) were procured and randomly distributed in to seven treatments, each treatment had six replicates with five chicks per each replicate. Growth trial was conducted in a randomized block design comprising seven dietary treatments. T1 was served as control. T2 and T3 were fed with garlic powder (GP) 0.5, 1 g/kg feed respectively. T4 and T5 were given with NLP 1, 2 g/kg feed respectively. T6 was fed with 0.5 g/kg GP and 1.0 g/kg NLP. T7 was treated with 1.0 g/kg GP and 2.0 g/kg NLP. The body weight, feed consumption of individual bird was recorded at weekly interval and the body weight gain, feed conversion ratio (FCR) were calculated. At the end of 42 day, six birds from each dietary group were sacrificed to estimate the dressing yield and relative weights of giblet (liver, heart and gizzard).Results: Supplementation of garlic and NLP alone and their combination significantly (p<0.05) improved the body weight gain and FCR whereas no significant (p>0.05) influence was observed in carcass parameters like dressing yield and giblet yield (heart, gizzard and liver) in all treatment groups.Conclusions: Supplementation of GP alone showed poor performance, but when it was supplemented in combination with NLP there was improvement in performance parameters. These results indicated that the combination of GP and NLP can be used as alternates to coccidiostats for improving the performance. Such meat can be used as a designer meat as fetch higher price if marketed as branded item.

In the present study, the effects of oral administration of imidacloprid for 4 weeks on serum biochemical, oxidative stress, histopathological and ultrastructural alterations were assessed in the liver of male rats. This study also aimed to investigate whether vitamin C could protect against the imidacloprid-induced oxidative stress. Forty-eight male Sprague dawley rats were divided into four groups of 12 animals each. Group 1 served as the control, while groups 2 and 4 were administered with imidacloprid (80 mg/kg body weight) daily by oral gavage for 28 days. In addition to imidacloprid, group 4 also received vitamin C at 10 mg/kg daily by oral gavage for 28 days. Group 3 was maintained as the vitamin C control (dose as above). The serum biochemical assays revealed a significant (P < 0.05) increase in alanine transaminase and aspartate transaminase and decrease in total protein in group 2. The tissue biochemical profile revealed a significant (P < 0.05) reduction in reduced glutathione concentration in the liver of group 2 animals. Histologically, the liver showed marked dilation, congestion of central vein, portal vein and sinusoidal spaces, vacuolation/fatty change and degenerated hepatocytes. Ultra thin sections of the liver revealed swollen nuclei, varied size and shape of mitochondria, disrupted chromatin and rough endoplasmic reticulum. Co-treatment with vitamin C significantly (P < 0.05) reversed the imidacloprid-induced changes.

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