A high frequency of latex sensitization and allergy was demonstrated in Hospital General de Mexico surgery residents, which indicates the need for policies and procedures to be developed for health workers with latex allergy, as well as continuous training of employees on latex allergy.
Los aditivos alimentarios son ingredientes agregados intencionalmente para Los aditivos alimentarios son ingredientes agregados intencionalmente para modificar las características físicas, químicas, biológicas o sensoriales de los alimentos; pueden actuar como alérgenos y generar reacciones inmunológicas mediadas por IgE o como pseudoalérgenos y generar reacciones inmunológicas no mediadas por IgE. Dichas reacciones pueden provocar diversos cuadros clínicos o exacerbar diferentes enfermedades: esofagitis eosinofílica, asma bronquial, dermatitis atópica, dermatitis de contacto, urticaria crónica o anafilaxia. Diariamente, miles de aditivos alimentarios son utilizados en países industrializados y aunque su uso se ha globalizado, se dispone de poca información científica sobre sus efectos adversos, particularmente sobre las reacciones de hipersensibilidad, las cuales son raras en la literatura, muy probablemente porque son infradiagnosticadas debido a que requieren un alto grado de sospecha clínica por parte del médico y la demostración de una relación de causalidad entre la sintomatología y el aditivo alimentario. Las reacciones de hipersensibilidad a aditivos alimentarios deben ser sospechadas en paciente con reporten síntomas a múltiples alimentos o a un alimento de preparación comercial sin sintomatología ante la ingesta natural o casera del mismo o ante reacciones idiopáticas. El papel diagnóstico de la prueba in vivo o in vitro contra IgE (pruebas cutáneas o RAST) está limitado a algunos aditivos alimentarios naturales. La prueba diagnóstica que demuestra causalidad entre el aditivo y la sintomatología es el reto oral alimentario. El tratamiento deberá ser siempre la eliminación del aditivo de la dieta.
The history of corticosteroids begins in the year of 1855 when Thomas Addison describes a "state of generalized languor, failure in the function of the heart, irritability in the stomach and changes of coloration in the skin", initially called melanodermia and later called Addison's disease, characterized by the lack of a substance produced in the adrenal glands [1].
Tolerance induction and desensitization in Stevens–Johnson syndrome (SJS) or in
toxic epidermal necrolysis (TEN) have been described as an absolute
contraindication by some authors, but there are cases where there is no
treatment alternative. Tuberculosis (TB) remains a leading cause of morbidity
and mortality in developing countries and ranks alongside HIV as a leading cause
of death worldwide. Severe drug reactions, such as SJS and TEN, occurring in
these individuals are lifethreatening. Since alternative therapies for TB are
limited, the role of desensitization and reintroduction becomes essential. We
describe a case of tolerance induction to anti-TB drugs in a patient with
SJS/TEN overlap syndrome using a specifically designed premedication,
comedication, and desensitization protocol.
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