The aim was to verify the validity (i.e., study A) and reliability (i.e., study B) of the alternative maximal accumulated oxygen deficit determined using onlya supramaximal effort (MAODALT)to estimate anaerobic capacity [i.e., estimated by the gold standard maximal accumulated oxygen deficit method (MAOD)] during cycling. In study A, the effects of supramaximal intensities on MAODALT and the comparison with the MAOD were investigated in fourteen active subjects (26 ± 6 years). In study B, the test-retest reliability was investigated, where fourteen male amateur cyclists (29 ± 5 years) performed the MAODALT twice at 115% of the intensity associated to maximal oxygen uptake (). MAODALT determined at 130 and 150% of was lower than MAOD (p ≤ 0.048), but no differences between MAODALT determined at 100, 105, 110, 115, 120 and 140% of (3.58 ± 0.53L; 3.58 ± 0.59L; 3.53 ± 0.52L; 3.48 ± 0.72L; 3.52 ± 0.61L and 3.46 ± 0.69L, respectively) with MAOD (3.99 ± 0.64L). The MAODALT determined from the intensities between 110 and 120% of presented the better agreement and concordance with MAOD. In the test-retest, the MAODALT was not different (p > 0.05), showed high reproducibility when expressed in absolute values (ICC = 0.96, p < 0.01), and a good level of agreement in the Bland-Altman plot analysis (mean differences ± CI95%:−0.16 ± 0.53L). Thus, the MAODALT seems to be valid and reliable to assess anaerobic capacity in cycling.
The purpose of the study was to investigate the sensitivity of an alternative maximal accumulated oxygen deficit (MAOD) method to discriminate the "anaerobic" capacity while comparing: least trained (LT) participants (n = 12), moderately trained (MT) participants (n = 12), endurance trained (ET) participants (n = 16), and rugby (RG) players (n = 11). Participants underwent a graded exercise test on a treadmill and a supramaximal effort for assessing MAOD. MAOD was calculated as the sum of oxygen equivalents from the phosphagen and glycolytic metabolic pathways. MAOD was significantly higher (P < 0.05) in RG (64.4 ± 12.1 mL · kg) than in ET (56.8 ± 5.4 mL · kg; effect size [ES] = 0.77; +13.5%), MT (53.8 ± 5.3 mL · kg; ES = 1.08; +19.8%), and LT (49.9 ± 4.5 mL · kg; ES = 1.50; +36.4%). In addition, the magnitude-based inference analysis revealed that MAOD was likely (LT vs. MT), very likely (MT vs. RG, and ET vs. RG) and most likely (LT vs. ET, and LT vs. RG) different between all groups, except for MT and ET, which presented an unclear difference. In conclusion, MAOD was sensitive enough to distinguish the "anaerobic" capacity in individuals with different training status, especially for RG players compared with LT participants and MT participants.
de Poli, RAB, Boullosa, DA, Malta, ES, Behm, D, Lopes, VHF, Barbieri, FA, and Zagatto, AM. Cycling performance enhancement after drop jumps may be attributed to postactivation potentiation and increased anaerobic capacity. J Strength Cond Res 34(9): 2465–2475, 2020—The study aimed to investigate the effects of drop jumps (DJs) on supramaximal cycling performance, anaerobic capacity (AC), electromyography, and fatigue. Thirty-eight recreational cyclists participated into 3 independent studies. In study 1 (n = 14), neuromuscular fatigue was assessed with the twitch interpolation technique. In study 2 (n = 16), the AC and metabolic contributions were measured with the maximal accumulated oxygen deficit method and the sum of the glycolytic and phosphagen pathways. In study 3 (n = 8), postactivation potentiation (PAP) induced by repeated DJs was evaluated. The DJ protocol was effective for significantly improving cycling performance by +9.8 and +7.4% in studies 1 and 2, respectively (p ≤ 0.05). No differences were observed in electromyography between conditions (p = 0.70); however, the force evoked by a doublet at low (10 Hz) and high frequencies (100 Hz) declined for control (−16.4 and −23.9%) and DJ protocols (−18.6 and −26.9%) (p < 0.01). Force decline was greater in the DJ condition (p < 0.03). Anaerobic capacity and glycolytic pathway contributions were +7.7 and +9.1% higher after DJ protocol (p = 0.01). Peak force during maximal voluntary contraction (+5.6%) and doublet evoked force at 100 Hz (+5.0%) were higher after DJs. The DJ protocol induced PAP, improved supramaximal cycling performance, and increased AC despite higher peripheral fatigue.
The aim of the present study was to investigate the differences in energy system contributions and temporal variables between offensive and all-round playing styles. Fifteen male table tennis players (Offensive players: N = 7; All-round players: N = 8) participated in the study. Matches were monitored by a portable gas analyzer and the blood lactate responses was also measured. The contributions of the oxidative (WOXID), phosphagen (WPCr), and glycolytic (W[La]) energy systems were assumed as the oxygen consumption measured during the matches above of baseline value, the fast component of excess post-exercise oxygen consumption (EPOCFAST) measured after the matches, and the net of blood lactate concentration (Δ[La]), respectively. Energy systems contributions were not significantly different between the offensive and all-round playing styles (WOXID: 96.1±2.0 and 97.0±0.6%, P = 0.86; WPCr: 2.7±1.7 and 2.0±0.6%, P = 0.13; W[La]: 1.2±0.5 and 1.0±0.7%, P = 0.95; respectively), however, magnitude-based analysis of WPCr presented Likely higher contribution for offensive compared to all-round players. Regarding temporal variables, only rate of shots presented higher values for offensive when compared to all-round players (P = 0.03), while the magnitude-based analysis presented Very likely lower, Likely lower and Likely higher outcomes of rate of shots, WPCr and maximal oxygen consumption, respectively, for all-round players. Strong negative correlation was verified for offensive players between number of shots and WPCr (r = -0.86, P = 0.01), while all-round players showed strong correlations between rally duration, WOXID (r = 0.76, P = 0.03) and maximal oxygen consumption (r = 0.81, P = 0.03). Therefore, despite no differences in energy system contributions for offensive and all-round players, different playing styles seems to requires specific energy systems demands.
The aim of the current study was to investigate the effects of acute caffeine supplementation on anaerobic capacity determined by the alternative maximal accumulated oxygen deficit (MAOD) in running effort. Eighteen recreational male runners [29 ± 7years; total body mass 72.1 ± 5.8 kg; height 176.0 ± 5.4cm; maximal oxygen uptake (VO) 55.8 ± 4.2 ml·kg ·min] underwent a graded exercise test. Caffeine (6 mg·kg) or a placebo were administered 1 hr before the supramaximal effort at 115% of the intensity associated with VO in a double-blind, randomized cross-over study, for MAOD assessment. The time to exhaustion under caffeine condition (130.2 ± 24.5s) was 11.3% higher (p = .01) than placebo condition (118.8 ± 24.9 s) and the qualitative inference for substantial changes showed a very likely positive effect (93%). The net participation of the oxidative phosphorylation pathway was significantly higher in the caffeine condition (p = .02) and showed a likely positive effect (90%) of 15.3% with caffeine supplementation. The time constant of abrupt decay of excess postexercise oxygen consumption (τ) was significantly different between caffeine and placebo conditions (p = .03) and showed a likely negative effect (90%), decreasing -8.0% with caffeine supplementation. The oxygen equivalents estimated from the glycolytic and phosphagen metabolic pathways showed a possibly positive effect (68%) and possibly negative effect (78%) in the qualitative inference with caffeine ingestion, respectively. However, the MAOD did not differ under the caffeine or placebo conditions (p = .68). Therefore, we can conclude that acute caffeine ingestion does not modify the MAOD, reinforcing the robustness of this method. However, caffeine ingestion can alter the glycolytic and phosphagen metabolic pathway contributions to MAOD.
The study investigated the influence of β-alanine supplementation during a high-intensity interval training (HIIT) program on repeated sprint ability (RSA) performance. This study was randomized, double-blinded, and placebo controlled. Eighteen men performed an incremental running test until exhaustion (TINC) at baseline and followed by 4-wk HIIT (10 × 1-min runs 90% maximal TINC velocity [1-min recovery]). Then, participants were randomized into two groups and performed a 6-wk HIIT associated with supplementation of 6.4 g/day of β-alanine (Gβ) or dextrose (placebo group; GP). Pre- and post-6-wk HIIT + supplementation, participants performed the following tests: 1) TINC; 2) supramaximal running test; and 3) 2 × 6 × 35-m sprints (RSA). Before and immediately after RSA, neuromuscular function was assessed by vertical jumps, maximal isometric voluntary contractions of knee extension, and neuromuscular electrical stimulations. Muscle biopsies were performed to determine muscle carnosine content, muscle buffering capacity in vitro (βmin vitro), and content of phosphofructokinase (PFK), monocarboxylate transporter 4 (MCT4), and hypoxia-inducible factor-1α (HIF-1α). Both groups showed a significant time effect for maximal oxygen uptake (Gβ: 6.2 ± 3.6% and GP: 6.5 ± 4.2%; P > 0.01); only Gβ showed a time effect for total (−3.0 ± 2.0%; P = 0.001) and best (−3.3 ± 3.0%; P = 0.03) RSA times. A group-by-time interaction was shown after HIIT + Supplementation for muscle carnosine (Gβ: 34.4 ± 2.3 mmol·kg−1·dm−1 and GP: 20.7 ± 3.0 mmol·kg−1·dm−1; P = 0.003) and neuromuscular voluntary activation after RSA (Gβ: 87.2 ± 3.3% and GP: 78.9 ± 12.4%; P = 0.02). No time effect or group-by-time interaction was shown for supramaximal running test performance, βm, and content of PFK, MCT4, and HIF-1α. In summary, β-alanine supplementation during HIIT increased muscle carnosine and attenuated neuromuscular fatigue, which may contribute to an enhancement of RSA performance. NEW & NOTEWORTHY β-Alanine supplementation during a high-intensity interval training program increased repeated sprint performance. The improvement of muscle carnosine content induced by β-alanine supplementation may have contributed to an attenuation of central fatigue during repeated sprint. Overall, β-alanine supplementation may be a useful dietary intervention to prevent fatigue.
To verify the acute effect of drop jumps (DJ) on two repeated sprint ability tests (RSA), interspersed with a rest period simulating a basketball game break. Twelve first division basketball players (age: 24.8 ± 6.9 years; body mass: 97.0 ± 9.2 kg; height: 2.0 ± 0.1 m) performed, in a randomized crossover design, two RSA tests separated by 5 min after DJ or control conditions. The DJ condition comprised 5 DJs performed 4 min prior to the first RSA test, whereas 3 DJ were completed 30 s prior to a second RSA test. Surface electromyography was recorded from the lower body for root mean square (RMS) analyses during sprinting. Three countermovement jump (CMJ) tests were performed after warming up and immediately after the second RSA test. DJ improved RSA performance with a faster best time in the first RSA test (p = 0.035), and a shorter total time and mean time (p = 0.030) for the second RSA test. No significant differences were found in RMS between protocols. CMJdecreased in both conditions after the RSA tests (p < 0.05). This study revealed a post-DJ RSA potentiation in professional male basketball players. This simple and effective approach could be implemented at the end of the warm-up and before the end of game breaks to improve player preparedness to compete.
The study aimed to identify and quantify the metabolites profile and metabolic pathways in human muscle tissue engaged during exhaustive high‐intensity cycling exercise. Seven healthy physically active men performed a graded exercise test and an exhaustive supramaximal effort at 115% of maximal aerobic power with muscles biopsies performed in rest and immediately after exhaustion for quantifying of muscle metabolites changes by 1H‐NMR spectroscopy. The time until exhaustion (tlim) recorded was 224.7 ± 35.5 s whereas the muscle pH at exhaustion was 6.48 ± 0.05. A total of 54 metabolites were identified and quantified. The most enriched and impacted pathways included: beta oxidation of very long chain fatty acids, mitochondrial electron transport chain, alanine aspartate, and glutamate metabolism, citric acid cycle, arginine biosynthesis, propanoate metabolism, threonine and 2‐oxobutanoate degradation and pyruvate metabolism. In addition, the muscle concentrations in Post exercise, compared to Pre increased significantly (p < 0.0398) for fumarate (42.0%), succinate (101.2%), glucose (249.7%), lactate (122.8%), O‐acetylcarnitine (164.7%), glycerol (79.3%), AMP (288.2%), 2‐oxobutyrate (121.0%), and methanol (58.5%), whereas decreased significantly (p < 0.010) for creatine phosphate (−70.2%), ADP (−56.5%), carnitine (−33.5%), and glutamate (−42.3%). Only the succinate was significantly correlated with tlim (r = −0.76; p = 0.0497). Besides the classical expected contribution of glycolytic and phosphagen energetic pathways, it was demonstrated that the high‐intensity exercise is also associated with pathways indicatives of amino acid and fatty acid oxidation metabolisms, highlighting the inverse relation between changes in the intramuscular succinate levels and tlim.
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