The erosion of lunar soil by rocket exhaust plumes is investigated experimentally. This has identified the diffusion-driven flow in the bulk of the sand as an important but previously unrecognized mechanism for erosion dynamics. It has also shown that slow regime cratering is governed by the recirculation of sand in the widening geometry of the crater. Scaling relationships and erosion mechanisms have been characterized in detail for the slow regime. The diffusion-driven flow occurs in both slow and fast regime cratering. Because diffusion-driven flow had been omitted from the lunar erosion theory and from the pressure cratering theory of the Apollo and Viking era, those theories cannot be entirely correct. IntroductionDuring the Apollo and Viking programs there was considerable research into the blast effects of launching and landing on planetary regoliths. That work ensured the success of those missions but also demonstrated that soil erosion or cratering will be a significant challenge for other mission scenarios. For example, the high-velocity spray of eroded soil will pose a serious challenge when we attempt to land multiple spacecraft within short distances of one another on the Moon. We have relevant experience because the Apollo 12 Lunar Module landed 155 meters away from the deactivated Surveyor 3 spacecraft. Portions of the Surveyor were returned by the Apollo astronauts to Earth for analysis. It was found that the surfaces had been sandblasted and pitted and that its openings had been injected with grit from the high-speed spray [Cour-Palais 1972]. This treatment is not acceptable for functional spacecraft.
The United States Department of Defense Blast Injury Research Program Coordinating Office organized the 2015 International State-of-the-Science meeting to explore links between blast-related head injury and the development of chronic traumatic encephalopathy (CTE). Before the meeting, the planning committee examined articles published between 2005 and October 2015 and prepared this literature review, which summarized broadly CTE research and addressed questions about the pathophysiological basis of CTE and its relationship to blast- and nonblast-related head injury. It served to inform participants objectively and help focus meeting discussion on identifying knowledge gaps and priority research areas. CTE is described generally as a progressive neurodegenerative disorder affecting persons exposed to head injury. Affected individuals have been participants primarily in contact sports and military personnel, some of whom were exposed to blast. The symptomatology of CTE overlaps with Alzheimer's disease and includes neurological and cognitive deficits, psychiatric and behavioral problems, and dementia. There are no validated diagnostic criteria, and neuropathological evidence of CTE has come exclusively from autopsy examination of subjects with histories of exposure to head injury. The perivascular accumulation of hyperphosphorylated tau (p-tau) at the depths of cortical sulci is thought to be unique to CTE and has been proposed as a diagnostic requirement, although the contribution of p-tau and other reported pathologies to the development of clinical symptoms of CTE are unknown. The literature on CTE is limited and is focused predominantly on head injuries unrelated to blast exposure (e.g., football players and boxers). In addition, comparative analyses of clinical case reports has been challenging because of small case numbers, selection biases, methodological differences, and lack of matched controls, particularly for blast-exposed individuals. Consequently, the existing literature is not sufficient to determine whether the development of CTE is associated with head injury frequency (e.g., single vs. multiple exposures) or head injury type (e.g., impact, nonimpact, blast-related). Moreover, the incidence and prevalence of CTE in at-risk populations is unknown. Future research priorities should include identifying additional risk factors, pursuing population-based longitudinal studies, and developing the ability to detect and diagnose CTE in living persons using validated criteria.
When a jet of gas impinges vertically on a granular bed and forms a crater, the grains may be moved by several different mechanisms: viscous erosion, diffused gas eruption, bearing capacity failure, and/or diffusion-driven shearing. The relative importance of these mechanisms depends upon the flow regime of the gas, the mechanical state of the granular material, and other physical parameters. Here we report research in two specific regimes: viscous erosion forming scour holes as a function of particle size and gravity; and bearing capacity failure forming deep transient craters as a function of soil compaction.Comment: 4 pages; Powders and Grains 2009, Golden, Colorado, US
scite is a Brooklyn-based organization that helps researchers better discover and understand research articles through Smart Citations–citations that display the context of the citation and describe whether the article provides supporting or contrasting evidence. scite is used by students and researchers from around the world and is funded in part by the National Science Foundation and the National Institute on Drug Abuse of the National Institutes of Health.
hi@scite.ai
10624 S. Eastern Ave., Ste. A-614
Henderson, NV 89052, USA
Copyright © 2024 scite LLC. All rights reserved.
Made with 💙 for researchers
Part of the Research Solutions Family.