The effects of chromium at 0.05,0.1,0.25,0.5 and 1.0 mM on iron, manganese, zinc and copper in maize (Zea mays L. cv.Ganga 5) were studied in controlled glass house conditions at ambient temperature after 56 days growth when plants started showing vein-clearing and papery appearence in the leaves.
Mango malformation is a major constrain in mango production worldwide causing heavy economic losses depending on cultivar type and susceptibility. The malady has variously been ascribed to be acarological, viral, fungal and physiological in nature. Here, we discuss the ethylene origin nature of malady. There are indications that most of the symptoms of mango malformation resemble with those of caused by ethylene effects. Multiple evidence reports of putative causal agents including Fusarium mangiferae to augment the endogenous pool of 'stress ethylene' are well documented. Therefore, over load of 'stress ethylene' impairs morphology malformed tissue and cyanide derived from ethylene biosynthesis causes necrosis and death of malformed cells. This review covers various factors eliciting 'stress ethylene' formation, role of ethylene in development of malady and regulation of ethylene action to reduce malformation in mango.
The concentration dependence of phosphate uptake was studied using root sections of corn (Zefl mays L. cv. Ganga 5). Detailed and widerange (57 concentrations in the range 1 /uniol m~^-75 mol m"-^), precise (average SEM <2.5%, n = 6) and reproducible (similar patterns in three independent experiments and for 5, 10, 15, 20, 25 and 30 °C) data revealed six (or seven) concentration-dependent phases separated by 'jumps' or sharp breaks. These transitions were independent of temperature and occurred over relatively narrow concentration ranges (0.0001-0.0004, 0.08-0.31, 1.0-3.5, (7.5-10), 18-20 and 57-59 mol m"-^). The intermediate phases obeyed Michaelis-Menten kinetics, whereas sigmoidal kinetics were observed at lower concentrations. Uptake within each of the two highest phases increased more rapidly with increasing external phosphate concentration than predicted from Michaelis-Menten kinetics but also saturated more rapidly. The latter finding is not consistent with free diffusion across the plasmalemma at high external phosphate concentrations. Kinetic models yielding continuous isotherms, e.g. the sum of one or two Michaelis-Menten terms and a diffusion term, cannot account for the data.
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