EMD) to determine EEG=EMG coupling. Results: Corticomuscular coupling was detected during the continually changing force levels. Different components included those over the primary motor cortex (ipsilaterally and contralaterally) and over the midline. Subjects with greater bradykinesia had a tendency towards increased $10 Hz coupling and reduced $30 Hz coupling that was erratically reversed with L-dopa. Conclusions: These results suggest that lower $10 Hz peak may represent pathological oscillations within the basal ganglia which may be a contributing factor to bradykinesia in PD.
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