References1 Yus ES, Requena L, Simon P, de Hijas CM. Incidental acantholysis. J Cutan Pathol 1993; 20: 418-423. 2 Yamamoto T. Dermatofibroma: a possible model of local fibrosis with epithelial/mesenchymal cell interaction. J Eur Acad Drmatol Venereol (accepted). 3 Morgan MB, Howard HG, Everett MA. Epithelial induction in dermatofibroma: a role for epidermal growth factor (EGF) receptor. Am J Dermatopathol 1997; 19: 35-40. 4 Han K-H, Huh C-H, Cho K-H. Proliferation and differentiation of the keratinocytes in hyperplastic epidermis overlying dermatofibroma: Immunohistochemical characterization. Am J Dermatopathol 2001; 23: 90-98. 5 Ackerman AB, Capland L, Rywlin AM. Focal keratosis follicularis overlying dermatofibroma. Br J Dermatol 1971; 84: 167-168.

The purpose of this study was to determine the frequency of contact allergy and photocontact allergy to sunscreens. A consecutive series of 54 patients with suspected clinical photosensitivity were assessed. All had the same standardized photobiological investigation from January 1989 to December 1990, including patch tests and photopatch tests with 6 sunscreen agents. Oxybenzone was found to cause 4 cases of allergic contact dermatitis (with photoaggravation in 2), and 3 cases of photocontact dermatitis (13% of patients). This is probably due to the wide distribution of oxybenzone in sunscreens and other cosmetics, 2 patients with polymorphic light eruption and persistent light reactivity, respectively, were regular sunscreen users. Photobiological investigation is necessary to ensure accurate diagnosis, since sunscreen contact or photocontact allergy may simulate other photosensitivity eruptions.

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