A series of 460 surgically treated cases of hydatid cyst of the lung is presented. Radiologic preoperative diagnosis was made in 90% of the patients. Cystotomy with subsequent capitonnage was performed in 395 cases, wedge resection in 27, lobectomy in 15 and segmental resection in 23 cases. After careful hemostasis, two drains were placed in the thorax and the thoracic cavity was closed. There were three postoperative deaths (0.6%) and two recurrences of cyst (0.4%) during follow-up investigation for periods up to 10 years.
Four patients with cystic hydatids located in pericardium and myocardium were admitted. Cardiac tamponade was determined in 1 of these cases. Surgical treatment was instituted for all cases. Transpericardial cardiac palpation was emphasized in cases where thoracotomy was necessary due to the lung cystic hydatids.
SUMMARYChylous ascites is a clinical entity characterized by accumulation of milky fluid containing high amounts of triglycerides in the peritoneal cavity. The cause is usually lymphatic obstruction secondary to neoplastic processes. Constrictive pericarditis rarely causes cylous ascites through elevated venous pressure and lymphatic stasis. To the best of our knowledge, there is no report of constrictive pericarditis leading to chylous ascites in a patient presenting with objective lymphangiographic findings of lymphatic obstruction rather than stasis. We present a case of chylous ascites and pleural effusion secondary to constrictive pericarditis presenting with signs of lymphatic obstruction in lymphangiography, in whom complete clinical and laboratory improvement was achieved after pericardiectomy. (Jpn Heart J 2004; 45: 535-540)
SUMMARYWe present two female nonsmoker Buerger's disease cases with anticardiolipin autoantibodies and a poor prognosis. One was a 64-year-old female who has had multiple lower and upper extremity amputations, while the other was a 32-year-old female with extremity and visceral artery involvement. Since both were positive for anticardiolipin antibodies, we speculate that Buerger's disease is an autoimmune disorder. (Int Heart J 2005; 46: 563-569) Key words: Buerger's disease, Anticardiolipin autoantibodies, Thromboangiitis obliterans BUERGER'S disease is an inflammatory thrombotic nonatherosclerotic vascular disease that affects arterial and venous vessels of medium and small caliber and the nerves within the neurovascular bundle. This vasculitis affects mainly the lower limbs and then the upper limbs, but has also been described in the visceral vessels. It principally strikes young men who are heavy smokers.1,2) It rarely affects women. Sasai, et al showed that nonsmokers at the first consultation were more common among women than among men. They also showed that clinical presentations in women with thromboangiitis obliterans (TAO) were almost equal to those in men.3)The etiopathogenesis is still obscure, but smoking appears to be tightly linked. 4) We think there are possible immunoallergic or autoimmune reactions with glycoprotein antigens in tobacco.The clinical criteria for diagnosis of TAO are a history of cigarette smoking, onset before age 50 years, infrapopliteal artery occlusive lesions, and the absence of atherosclerotic risk factors other than smoking. Arteriographic findings include tapering or abrupt occlusion of peripheral arterial vessels, a corkscrew or From the
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