We consider a system of delay differential equations to represent predator-prey eco-epidemic dynamics with weak Allee effect in the growth of predator population. The basic aim of the paper is to observe the dynamics of such system under the influence of gestation delay of predator and Allee parameter. We analyze essential mathematical features of the proposed model such as uniform persistence, stability and Hopf-bifurcation at the interior equilibrium point of the system. Global asymptotic stability analysis of the positive equilibrium points by constructing a suitable Lyapunov function for the delayed model is carried out separately. We perform several numerical simulations to illustrate the applicability of the proposed mathematical model and our analytical findings. We observe that the system exhibits chaotic oscillation due to increase of the delay parameter τ. We also observe that there is a threshold of Allee parameter above which the predator population will be washed away from the system.
Cellular Vehicle-to-everything (C-V2X) communication is a major V2X solution proposed and developed by the 3rd Generation Partnership Project (3GPP). Our previous work has studied scalability aspects of C-V2X and demonstrated its potential for accommodating large numbers of vehicles in dense vehicular scenarios. However, existing studies in the scientific literature mostly have a network-level approach to the problem and do not assess the temporal and spatial dynamics of C-V2X networks in heavy network load situations. In this work we shed light on the spatio-temporal characteristics of these networks and investigate the effectiveness of the congestion control algorithm in dense vehicular ad-hoc networks (VANETs) in terms of settling time, stability, and reliability to be employed for the purpose of safety-critical vehicular applications, where latency plays a major role.
The paper explores an eco-epidemiological model of a predator-prey type, where the prey population is subject to infection. The model is basically a combination of S-I type model and a Rosenzweig-MacArthur predator-prey model. The novelty of this contribution is to consider different competition coefficients within the prey population, which leads to the emergent carrying capacity. We explicitly separate the competition between non-infected and infected individuals. This emergent carrying capacity is markedly different to the explicit carrying capacities that have been considered in many ecoepidemiological models. We observed that different intra-class and inter-class competition can facilitate the coexistence of susceptible prey-infected prey-predator, which is impossible for the case of the explicit carrying capacity model. We also show that these findings are closely associated with bi-stability. The present system undergoes bi-stability in two different scenarios: (a) bi-stability between the planner equilibria where susceptible prey co-exists with predator or infected prey and (b) bi-stability between co-existence equilibrium and the planner equilibrium where susceptible prey coexists with infected prey; have been discussed. The conditions for which the system is to be permanent and the global stability of the system around disease-free equilibrium are worked out.
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