This pilot project shows that it is feasible and valuable to screen patients with TB for DM in a routine setting, resulting in earlier identification of DM and opportunities for better management of comorbidity.
Introduction: Diabetes mellitus is one of the major noncommunicable diseases of which world is experiencing a serious epidemic these recent years. Uric acid serves as an early indicator of renal complications in diabetes mellitus patients.
Material and methods:This was an Observational, descriptive cross sectional study which was conducted during a period of 18 months (October 2016 and March 2018) This study was designed to check the levels of serum uric acid and its relation with creatinine, microalbuminuria, HbA1c, fasting and post prandial blood sugar levels in type 2 Diabetes subjects.Results: Study included a total of 120 cases of type 2 Diabetes mellitus, out of which there were 69 males (57.5%) and 51 females (42.5%), with a mean age of 59.04 ±13.47 years. Mean FBS was 186.10 ±77.53 mg/dl, with majority of the subjects having elevated FBS. Mean PPBS of 274.94 ±108.66 mg/dl and of HbA1c 8.15 ± 1.7 was observed. The uric acid of majority number of our study participant males (65.22%) had level of ≥ 7.4, with a mean of 9.53 ± 4.38. Mean blood urea and serum creatinine levels were 46.91 ± 15.13 and 1.44 ± 0.29 respectively. There was significant association seen between uric acid levels and urine albumin, serum creatinine, twenty four hour urinary albumin, FBS and PPBS levels and HbA1c levels.
Conclusion:Present study had about two-third subjects with type 2 Diabetes mellitus with elevated uric acid levels had microalbuminuria and elevated serum creatinine levels.
Cardiac and renal diseases are becoming increasingly common today, and are seen to frequently coexist, thus causing a significant increase in the mortality rate, morbidity, complexity of treatment and cost of care. Syndromes describing the interaction between heart and kidney have been defined and classified; however, never as a result of a consensus process. Though the incidence of cardiorenal syndrome is increasing, the associated pathophysiology and effective management are still not well understood. For many years, diuretics and ultrafiltration, have been the mainstay of treatment for cardiorenal syndrome, although a significant proportion of patients develop resistance to diuretics, and even deteriorate while on diuretics. Here, we will discuss one such patient who failed to respond to the optimum doses of diuretics; however, his blood urea and serum creatinine touched the baseline levels post-ultrafiltration.
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