Two studies were conducted to determine whether adding exercise to a diet programme promotes weight loss or glycaemic control in Type 2 (non-insulin-dependent) diabetic subjects. In Study 1, 25 subjects were randomly assigned to diet plus moderate exercise or diet plus placebo exercise. All subjects exercised twice a week as a group and once a week on their own; the diet plus moderate exercise group walked a 3-mile route at each session while the diet plus placebo exercise group did very low intensity exercises such as stretching and light calisthenics. All subjects followed a calorie-counting diet and were taught behaviour modification strategies. Weight losses and improvements in glycaemic control did not differ significantly between the two treatment groups at the end of the 10-week treatment or at 1-year follow-up. In Study 2, more extreme conditions were compared: a diet only group and a diet plus exercise group. The diet plus exercise group walked a 3-mile route with the group 3 times/week and once a week on their own, while the diet only group was instructed to maintain their current low level of activity. Both groups received comparable diet and behaviour modification instruction and therapist contacts. The diet plus exercise group had significantly (p less than 0.01) better weight losses than the diet only condition at the end of the 10 week programme (-9.3 kg vs -5.6 kg) and at 1 year follow-up (-7.9 kg vs -3.8 kg).(ABSTRACT TRUNCATED AT 250 WORDS)
Imbalance of the eccentrically-activated external rotator cuff muscles versus the concentrically-activated internal rotator cuff muscles is a primary risk factor for glenohumeral joint injuries in overhead activity athletes. Nonisokinetic dynamometer based strength training studies, however, have focused exclusively on resulting concentric instead of applicable eccentric strength gains of the external rotator cuff muscles. Furthermore, previous strength training studies did not result in a reduction in glenoumeral joint muscle imbalance, thereby suggesting that currently used shoulder strength training programs do not effectively reduce the risk of shoulder injury to the overhead activity athlete. Two collegiate women tennis teams, consisting of 12 women, participated in this study throughout their preseason training. One team (n = 6) participated in a 5-week, 4 times a week, external shoulder rotator muscle strength training program next to their preseason tennis training. The other team (n = 6) participated in a comparable preseason tennis training program, but did not conduct any upper body strength training. Effects of this strength training program were evaluated by comparing pre- and posttraining data of 5 maximal eccentric external immediately followed by concentric internal contractions on a Kin-Com isokinetic dynamometer (Chattecx Corp., Hixson, Tennessee). Overall, the shoulder strength training program significantly increased eccentric external total work without significant effects on concentric internal total work, concentric internal mean peak force, or eccentric external mean peak force. In conclusion, by increasing the eccentric external total exercise capacity without a subsequent increase in the concentric internal total exercise capacity, this strength training program potentially decreases shoulder rotator muscle imbalances and the risk for shoulder injuries to overhead activity athletes.
Little is known about the relation between serum sex hormones and either coronary heart disease or the development of atherosclerosis in women. We measured serum estrone concentrations in 87 postmenopausal women (age, 50 to 81 years) who were admitted for diagnostic cardiac catheterization. None of the women were on estrogen replacement therapy. Cases (n=62) were defined as those women who had si coronary artery with £50% occlusion. All control subjects (n=25) had 0% to 24% occlusion of all coronary arteries. Estrone concentrations, as measured by a combination of extraction, column chromatography, and radioimmunoassay, showed little difference between cases and control subjects. A difference of 6 pg/mL in the estrone level was not associated with a significantly increased risk of coronary artery disease (odds ratio [OR], 1.85; 95% confidence intervals [CI], 0.60, 5.2). Examination of mean estrone levels on the basis of the number of occluded vessels was also not significant. The primary predictors of coronary artery disease in this population were a history of diabetes (OR, 8.8; CI, 1.5, 51.4) and age (5-year increments; OR, 2.1; CI, 1.2, 3.8). There was also some suggestion that women who reported higher lifetime physical activity levels were at a reduced risk for developing coronary artery disease (OR, 0.18; CI, 0.05, 0.65). These preliminary results do not support the hypothesis that serum estrogens are related to coronary artery disease in older women, but these findings need to be replicated in larger populations of older women. (ArterioscUr Thromb. 1994 There are approximately 250 000 deaths in women due to heart disease each year in the United States. The observations of a predominance of CHD among men, the diminishing differences in CHD rates in males versus females with increasing age, comparison of the risk of CHD across menopause, data on the use of oral contraceptives and noncontraceptive estrogens, and the risk of CHD strongly suggest that hormonal factors, specifically sex hormones, may play a major role in the development of CHD. The relation of serum sex hormones to both fatal and nonfatal CHD 2 and to the extent of atherosclerosis 3 has been examined in men. In contrast, little is known about the relation of serum sex hormones to CHD or to the development of atherosclerosis in women.There is a very substantial increase in the risk of both CHD 4 and atherosclerosis 5 among women after surgical menopause. For natural menopause, mortality data from the United Kingdom 6 and the United States 7 do not show an abrupt change in mortality rates near the age of menopause. However, data from the Framingham Study suggest that female CHD morbidity rates accelerate more quickly than do those of males after age 45. There has also been considerable research on the relation of exogenous estrogens to CHD in women. On the basis of a critical review and meta-analysis of all epidemiological data, Bush 9 has estimated that long-term use of postmenopausal estrogen decreases the risk of cardiovascular diseas...
Life-style activities such as walking are often recommended for patients with type II (non-insulin-dependent) diabetes. Because many of these patients are overweight and sedentary, such low-intensity activity would appear most appropriate, especially during initial intervention. However, there has been little research on the effects of low-intensity life-style activity on glycemic control. This study examined the effects of varying the duration (0, 20, or 40 min) of low-intensity exercise (50-55% of age-predicted max heart rate) on glycemic responses during exercise and a subsequent meal in type II diabetic patients. Glycemic response to exercise was significantly related to the duration of activity; 20 min of activity decreased blood glucose (BG) by 6 mg/dl, whereas 40 min decreased BG by 16 mg/dl. The effect of exercise on glucose was maintained over a 30-min rest period but disappeared after a meal was consumed. Insulin and the insulin-to-glucose ratio were not affected by the length of activity. These data suggest that life-style activity of long duration (20-40 min) produces a significant, but modest, decrease in glucose levels in type II diabetic women.
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