Encapsulated collections of bile ("biloma") may be a sequela of liver trauma, operative injury or disease. Such collections may be intrahepatic or extrahepatic and usually in the supramesocolic compartment of the abdomen. This is a report of a retroperitoneal biloma, an entity that has been reported only twice to date but this is the first secondary to an operative common bile duct lesion.Evacuation of the biloma and reconstruction of the associated biliary stricture were successfully carried out. The patient remains sympton free with normal clinical and laboratory data more than 14 months after surgery.Operative common bile duct (CBD) injury may be followed by a number of complications. To our knowledge retroperitoneal biloma secondary to a CBD lesion has not been previously reported.
CASE REPORTA 54 year old lady was submitted to elective cholecystectomy in a district hospital on 15/09/1988. Operative cholangiography was not performed. Soon after operation bile drainage through a subhepatic drain appeared and continued for four weeks. The patient subsequently developed bile peritonitis and was reoperated upon on 21/10/1988 and the abdominal cavity was drained. Bile drainage stopped by mid-January 1989 but was followed by jaundice. A third operation was performed on 23/02/89 for obstructive jaundice. The CBD could not be found and the abdomen was closed with drainage. In March 1989 percutaneous transhepatic cholangiography (PTC) was carried out clearly demonstrating a high CBD stricture. The procedure was followed by frequent attacks of severe cholangitis with a temperature of up to 40C.. Once while vomiting she felt "that something broke in the abdomen". After this episode the fever abated somewhat and the attacks of cholangitis became less severe. However, a painful enlarging right lower quadrant abdominal mass appeared.At the time of admission to our Institution on 31/03/1989 she was deeply jaundicied with a large tender right lower abdominal mass. Laboratory data indicated biliary obstruction (total serum bilirubin 302.5 mmol/l, serum alkaline phosphatase 6.9 U/I (normal value 1 to 2.5 U/l).
The aim of this study is the assessment of the relative arterial and venous contribution to the total liver blood flow (hepatic perfusion index-HPI), with two methods (S1 and S2), and estimation of their value. With this correction, HPI nonsignificantly increases (p>0.05) in all the groups of patients, with a very high correlation between the HPI (S1) and HPI (S2) values (p<0.01). In comparison to the portal perfusion in controls, values were significantly (p<0.01) lower in chronic active hepatitis and liver cirrhosis and differed between themselves (p<0.01). In the groups of cirrhotic patients with esophageal varices, sclerosated esophageal varices, recanalized umbilical vein, portal thrombosis and cavernous portal vein, portal perfusion was lower (p<0.01) than in controls, chronic active hepatitis and liver cirrhosis without collaterals. Both angioscintigraphic methods are useful for the estimation of the disturbances in the portal system. Because of the more exact estimation of the liver perfusion, S2 is recommended.
Inflammatory pseudotumors are very rare benign lesions of an unknown etiology usually discovered accidentally. We present a 26-year-old woman with a nodal lesion discovered in the spleen during a routine check-up by abdominal ultrasound. Radiocolloid and delayed Tc-99m RBC scintigraphy proved the existence of a delineated hypervascular lesion. Histology and immunohistochemistry of the lesion proved an inflammatory pseudotumor, with very dilated sinusoids with blood congestion. The exact diagnosis of inflammatory pseudotumor can be established only by histology and/or immunohistochemistry of the spleen.
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