Exposure to specific spatial-temporal regulated electromagnetic field configurations had potent effects on the growth of experimental tumours in mice.

The results suggest that a common factor in cellular adhesion or plasticity might be permanently altered by prenatal exposure to a narrow intensity of a series of physiologically-patterned magnetic fields.

No abstract

The brains of adult rats, exposed prenatally to one of four intensities (between 10 nanoTesla and 1.2 microTesla) of either a frequency-modulated magnetic field or a complex sequenced field designed to affect brain development, were examined histologically. Although from each intensity some rats that had been exposed to the complex sequenced magnetic field showed minor anomalies, those exposed to intensities between 30 nT and 180 nT exhibited conspicuous anomalous organizations of cells within the hippocampal formation. In other studies, rats that had been exposed during their entire prenatal development to the complex sequenced field displayed significantly more activity in the open field and poorer spatial memory during maze learning. Photomicrographs are shown of one conspicuous morphological anomaly within the right hippocampus of an adult rat exposed prenatally to the complex sequenced magnetic field with intensities between .3 mG and .5 mG (30 nT to 50 nT). The results suggest that complex magnetic fields, whose temporal structures approach the time constants of normal biochemical processes, can permanently alter the development of the brain.

Exposure to sinusoidal (power-frequency) magnetic fields during prenatal development is implicated in adulthood behavioral impairments. However, the effects of prenatal exposure to weak-intensity, nonsinusoidal complex magnetic fields (CMFs), an increasingly common feature of the modern environment, have not been rigorously examined. In the present study, male and female Wistar-strain rats were exposed continually during prenatal development to one of three extremely low-frequency CMFs or a sham condition. As adults, rats were trained in an acquisition/reversal radial maze task. All rats exposed to the prenatal CMFs increased their commission of reference memory errors, but differences in working memory and motivation to complete the maze task were specific to the type of prenatal CMF. These results provide the first evidence that prenatal exposures to specific shapes of CMFs impair complex learning behaviors into adulthood.