Objective: To determine the relationship between symphysis dimensions and alveolar bone thickness (ABT) of the mandibular anterior teeth. Methods: Cone-beam computed tomography images of 51 patients were collected and measured. The buccal and lingual ABT of the mandibular anterior teeth was measured at 3 and 6 mm apical to the cemento-enamel junction (CEJ) and at the root apices. The symphysis height and width were measured. The symphysis ratio was the ratio of symphysis height to symphysis width. Kendall’s tau correlation coefficient was used to determine the relationships between the variables at a 0.05 significance level.Results: The mandibular anterior teeth lingual and apical ABT positively correlated with symphysis width (p<0.05). Moreover, these thicknesses negatively correlated with the symphysis ratio (p<0.05). Symphysis widths and ratios showed higher correlation coefficients with total and buccal apical ABT, compared with lingual ABT. Buccal ABT at 3 and 6 mm apical to the CEJ was not significantly correlated with most symphysis dimensions. The mean thickness of the buccal alveolar bone at the upper root half was only 0.2-0.6 mm, which was very thin, when compared with other regions. Conclusion: For mandibular anterior teeth, the apical alveolar bone and lingual alveolar bone tended to be thicker in patients with a wide and short symphysis, compared to those with a narrow and long symphysis. Buccal alveolar bone was, in general, very thin and did not show a significant relationship with most symphysis dimensions.
There are more compressive stresses concentrated at the apex of incisors with a high degree of inclination than in incisors that are more upright. This may be associated with the higher clinical incidence of apical root resorption found in inclined maxillary central incisors.
Amelogenesis imperfecta (AI) refers to a group of rare genetic disorders that involve tooth development and that are passed down through families as a dominant trait. This condition is characterized by abnormal enamel formation caused by gene mutations that alter the quality and/or quantity of enamel. This dental problem can impact both primary and permanent dentition, varies among affected individuals, and results in esthetic and functional problems. This condition caused the patient in the current case report to have a lack of confidence when speaking. The treatment for amelogenesis imperfecta depends on the severity of the problem and age of the patient. It is crucial to plan a proper remedy, which requires collaboration among dental specialties to execute comprehensive dental treatment in order to provide a long-term solution with adequate esthetics. The current clinical study presents a patient affected by AI that was diagnosed when the patient was a child. The interdisciplinary treatment continued throughout his childhood and into adult life. The initial treatment consisted of resin composite veneers and stainless-steel crowns to restore the defective tooth structure. The malocclusion of the patient was corrected using a fixed orthodontic appliance that was placed when he had an entire permanent dentition. The treatment plan was eventually intended to include all ceramic crowns and veneers.
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