Anemic children were 5.75 times more susceptible to LRTI compared to the control group. Prevention of anemia, due to whatever etiology, will reduce the incidence of LRTI.

Objective:This prospective-(cohort) study was conducted to evaluate whether anemia is a risk factor for childhood asthma.Materials and Methods:Two hundred children in the age group of 2-18 years who attended the Outpatient Department with upper respiratory / lower respiratory tract infections were included in this study. One hundred children with anemia were taken as the study group and another 100, age - and sex-matched children without anemia were taken as the control.They were subjected to complete blood count (CBC) C-reactive protein (CRP) estimation, Mantoux test and chest X-ray. Pulmonary function tests (PFTs) were performed on those above six years showing evidence of asthma. Peripheral smear, serum ferritin and serum iron-binding capacity were estimated for all anemic children.Results:Asthma was present in 74 (74%) children in the study group and in 33 (33%) children in the control group. Iron-deficiency anemia was present in 85 (85%) anemia of chronic infection in 20 (20%) and the other five (5%) had hemolytic anemia. Anemia was found to be a risk factor for childhood asthma.Conclusion:Anemic children were 5.75 times more susceptible to asthmatic attacks when compared with nonanemic children.

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We have documented an overall predominance of right hepatic lobe metastases independent of site of the primary colorectal carcinoma. However, when metastases occurred in the left lobe, the left lateral segment (II, III) was more commonly affected than the medial segment (IV), but this difference was statistically significant only when there were four or less segments involved with liver metastases.

We report a 30-day-old baby with subcutaneous fat necrosis and symptomatic hypercalcemia, who developed metastatic calcification in the subcutaneous tissue, kidneys, pericardium and brain. The baby also had anemia, hypertriglyceridemia and hypercholesterolemia. He was managed with intravenous saline, furosemide, oral steroids and bisphosphonates and improved with treatment.