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The objective of this investigation is to clearly understand the aerodynamic characteristics of a small-sized wind turbine of NREL Phase VI, operating with a stall-regulated method using CFD code. Based on this, it is possible to provide turbine designers with the aerodynamic design data to increase efficiency and improve performance in the design phase of future small-sized wind turbine blades. Moreover, a comparison was made between experimental datasets, in order to verify the reliability and validity of the analysis results. The first height in the normal direction from the surface of a rotor blade is about 0.2 mm, and the average value of y + is about 7 at 7 m/s. The domain is chosen to consist of only two hexahedral mesh regions, namely the interior region, including the wind turbine blade, and the external region excluding the rectangle. The total cell number of the numerical grid is about N g = 3.0 × 10 6 . Five different inflow velocities, in the range V in = 7.0-25.1 m/s, are used for the rotor blade calculations. The calculated power coefficient is about 0.35 at a TSR of 5.41, corresponding to 7 m/s, and showed considerably good agreement with the experimental measurements, to within 0.08%. It was observed that the 3-D stall begins to generate near the blade root at a wind speed of 7 m/s. Therefore, root design approaches considering the appropriate selection of the angle of attack and the thickness are very important in order to generate the stall on the blade root. Through a clear understanding of aerodynamic characteristics of a small-sized NREL Phase VI wind turbine, it is expected that this useful aerodynamic data will be made available to designers as guidance in designing stall-regulated wind turbine blades in the development phase of small-sized wind turbine systems in the future.

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Large Eddy Simulation (LES) of the NREL (National Renewable Energy Laboratory) Phase VI wind turbine inside a virtual wind tunnel, with the same test section as that of NASA Ames 24.4 m × 36.6 m, was carried out in order to analyse and better understand the wake instability and its breakdown behind the wind turbine. LES was performed using the commercial CFD software, ANSYS FLUENT, based on the dynamic Smagorinsky-Lilly model. The wind turbine was placed at a distance of two rotor diameters from the upstream boundary with a downstream domain of twenty rotor diameters in length The results of the simulation were compared with the experimental data published by the NREL and a good agreement was found between the two. Furthermore, the average turbulence intensities from the LES were compared with a semi-empirical model and very good agreement was observed, except for the regions of on-going wake instability and vortex breakdown. It was observed that the wake behind the wind turbine consists of a system of intense and stable rotating helical vortices.These vortices persisted for some distance downstream of the wind turbine and finally become unstable producing a sinuous shape. The downstream distance at which wake instability and vortex breakdown occur, was observed to be a function of the upstream wind speed. For example, for an upstream wind speed of 7 m/s, it was observed that the primary vortex structure became unstable at a downstream distance of four rotor diameters and complete breakdown occurred at approximately six rotor diameters. On the other hand, when the upstream wind speed was 15.1 m/s, wake instability occurred at approximately eleven rotor diameters downstream of the wind turbine and complete breakdown was observed at thirteen rotor diameters downstream of the wind turbine. Furthermore, it was observed that the turbulence intensity rapidly decreased during the process of wake instability and vortex breakdown; the location of the decrease is a function of the upstream wind speed. It is suggested that the distinction between the near and far wake can be identified as the average location between the start of the wake instability and the end of the process, at complete breakdown. Therefore the average location of this boundary is a function of the upstream wind speed. Hence for upstream wind speeds of 7 m/s, 10 m/s, 13.1 m/s and 15.1 m/s, the boundary between the near and far wake lies at five, seven, ten and twelve rotor diameters downstream respectively.

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Bile reflux contributes a lot to mucosal lesions in the whole stomach, may facilitate H pylori colonization in the corpus region, and has no influence on acid-exposing status of gastric mucosa in patients with dyspepsia or chronic gastritis.

Converging lines of evidence demonstrate a vagally mediated antinociceptive pathway in animals undergoing acute visceral insults, the contribution of this system to visceral pain following chronic noxious stimuli is unknown. 5-HT(3) receptor (5-HT(3)Rs) on spinal afferents are crucially involved in nociceptive processing, the role of 5-HT(3)Rs on vagal afferents is unclear. The aim of the present study was to determine the contribution of vagal afferents to visceral nociception in rats undergoing chronic luminal allergen stimulation and whether it involves vagal 5-HT(3)Rs. Sensitized rats received chicken egg albumin (EA, 1 mg mL(-1)) in drinking water for 2 weeks (day 1-14). Visceromotor response (VMR) to colorectal distension [colorectal distension (CRD), 60 mmHg] and the levels of mRNA encoding 5-HT(3)R (including 3A and 3B subunits) in the nodose ganglia (NG) were evaluated on day 2, 4, 8 and 15. Chronic EA challenge induced gradually increased visceral nociception, with a peak on day 15. Subdiaphragmatic vagotomy or functional deafferentation with capsaicin abolished this time-dependent manner, inducing hyperalgesia from day 2, lasting to day 15. Intraluminal infusion of a 5-HT(3)R antagonist (granisetron), whether alone or infused after local mucosa anaesthetic with 1% lidocaine, mimicked the effects of vagotomy. The mRNA levels for 5-HT(3B) or 5-HT(3A) subunit in the NG showed an opposite time-course to that of visceral pain, which increased from day 2, then decreased gradually to levels lower than those of controls. Our results demonstrate a time-dependent vagal afferent modulation of chronic allergen-sensitized visceral hyperalgesia, which may involve a 5-HT(3)R pathway.

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