These findings may be useful in the clinical setting and point to heterogeneity of AD according to age at onset.

OBJECTIVE: To assess the role of impaired encoding in learning and in delayed recall disturbances, and to evaluate the rate of forgetting in AD. METHOD: Fifteen AD patients with mild or moderate dementia and 15 normal matched controls were assessed with the Buschke Selective Reminding Test. Delayed recall was evaluated after 30 minutes and after 24 hours. RESULTS: AD patients had a poorer performance across the six trials of the learning phase as well as in both delayed recall evaluations, with no difference between recall at 30 minutes and at 24 hours. CONCLUSION: Performance in the learning phase was as specific and almost as sensitive as the performance in delayed recall for AD diagnosis. Encoding impairment was responsible for poorer learning and rapid displacement of previous learned material in the AD group. Finally, we did not find a higher rate of forgetting in AD patients.

This report describes the case of a 39-year-old male patient who presented to the emergency room with complaints of impaired balance, diplopia, and nasal voice. The patient had a history of upper respiratory tract infection. The initial physical examination revealed ataxia, ophthalmoplegia, and areflexia, which are consistent with the classic triad of Miller Fisher syndrome, considered a benign variant of Guillain-Barré syndrome. The patient developed peripheral facial paralysis during hospitalization. He underwent a treatment with immunoglobulin for five days, resulting in near complete resolution of the ataxia. However, the ophthalmoplegia and areflexia persisted. He was discharged to outpatient follow-up.

A doença de Lyme é causada pela infecção por diferentes espécies do gênero Borrelia e transmitida por picada de carrapatos da família Ixodidae e similares sendo responsável por manifestações clínicas sistêmicas como cutâneas, articulares, cardíacas e neurológicas. Por apresentar particularidades quanto ao agente etiológico, transmissão e diagnóstico, e por não existirem no Brasil relatos conclusivos da ocorrência da Doença de Lyme com as características clínico-laboratoriais encontradas no hemisfério norte, foi optado pela mudança da nomenclatura no Brasil para Síndrome de Baggio-Yoshinari (SBY). É analisado nesse relato um caso internado no Hospital Universitário da Universidade de São Paulo que cursou exclusivamente com manifestações neurológicas da SBY (radiculite periférica, paralisia facial e comprometimento cerebelar) e ausência de manifestações cutâneas ou articulares.