A mass that developed on the shoulder of a 65‐year‐old woman was diagnosed histologically as a keratotic basal‐cell carcinoma arising from the wall of an epidermoid cyst.
Multiple cystic and proliferative follicular lesions localized on the face and the genitalia of several members of an Italian family are described. Transmission seems to be autosomal dominant, with weak penetration and variable expressivity.
Summary
Since 1969, 79 cases of fungal maxillary sinusitis have been diagnosed. Forty-nine were due to Aspergillus fumigatus. There were no underlying diseases which depressed cellular immunity and no patient was receiving immunosuppressive drugs or corticosteroids. Most patients had received antibacterial therapy before the appearance of FMS. Treatment was by surgery, nystatin and econazole.
Three infants, 4 to 9 months old, treated for ‘seborrheic dermatitis’ with topical fluorinated corticosteroids present clinically nodular and histologically granulomatous lesions in the genito-inguinal regions and over the buttocks; mycological investigations and PAS stains of tissu sections show the presence of yeast elements identified as being Candida albicans. No general illness favoring the development of granulomatous candidiasis could be found. This fact together with the location of the eruption permit to suppose that the lesions are due to the application of fluorinated steroids, the treated region being covered with plastic pants over the diapers.
Since 1969, 600 patients suffering from maxillary sinusitis have been systematically controlled clinically as well as mycologically. We discovered 81 cases of maxillary sinusitis (13,4%). Aspergillus fumigatus and Candida albicans were the most frequently isolated fungi. Three clinical forms could be distinguished: (1) a muco-purulent form (35 cases); (2) a budding and/or caseous form (22 cases), and (3) a pseudotumoral form (24 cases). 42 patients treated with a 1 % econazol (base) solution were healed.
38 biopsies of perioral dermatitis have been examined. The following has consistently been observed: follicular hyperkeratosis; vasodilatation and oedema (sometimes very pronounced) of the papillary dermis; peri vascular and parafollicular infiltrates consisting predominantly of lymphocytes, histiocytes and polymorphonuclear leucocytes. Occasionally, there have been epithelioid or giant cells, sarcoid-like infiltrates or vasculitis of the leucoclastic type. Demodexfolliculorum was observed in only two sections and the follicles that contained them were not inflamed. This histological picture is very similar to that of acne rosacea.
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